目的探讨早期内毒素休克肾脏损伤的发病机制。
OBJECTIVE: To study the pathogenesis of kidney injury in early phase of endotoxic shock.
虎杖甙对内毒素休克大鼠肺组织中磷脂酶a2的影响。
Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock.
目的了解内毒素休克后血管反应性的变化及器官差异。
Objective to explore the changes and organ diversity of vascular reactivity following endotoxic shock.
建立D -氨基半乳糖敏化小鼠内毒素休克的规范化模型。
To establish the standard model of endotoxins shock of mice sensitized with D-galactosamine.
目的：探讨内毒素休克后大鼠肠粘膜免疫功能的改变及意义。
Objective: To investigate the changes of gut mucosal immune function in rats with endotoxic shock and its significance.
这也是补充蛋白C是治疗内毒素休克一项极具希望疗法的所在。
That is probably also why protein C supplementation is such a promising potential therapy for septic shock.
目的：研究钙增敏剂mci - 154对内毒素休克家兔心功能的影响。
Objective: to observe the effects of MCI 154, a calcium sensitizer, on cardiac function after endotoxic shock.
目的了解内毒素休克后血管反应性的变化规律及其与血流动力学变化的关系。
Objective To explore the changes of vascular reactivity following endotoxic shock and its relation with hemodynamic changes.
目的：研究钙增敏剂mci - 154对内毒素休克大鼠心肌钙稳态的影响。
Objective: to investigate the effects of MCI 154, a calcium sensitizer, on myocardial calcium homeostasis during endotoxic shock in rats.
方法：观察双黄连注射液对内毒素休克小鼠的生存时间及对内毒素所致大鼠肝、肺、肾等病理改变的影响。
METHODS To observe the effect of Shuanghuanglian injection on the average death time of mice and the pathological changes of rats livers , lungs and kidneys induced by endotoxin.
结论内毒素休克血管反应性呈现一定器官差异，这种差异可能与内毒素休克血流动力学改变及血液重分布有关。
Conclusion There is organ diversity of vascular reactivity following endotoxic shock, which may be related to the changes of hemodynamics and blood redistribution.
结论：钙增敏剂可逆转内毒素休克所致的心肌收缩蛋白对钙反应性的降低，改善内毒素休克机体的心肌收缩性能。
Conclusion: Calcium sensitizer can reverse the reduced responsiveness of myocardium to Ca2 + and improve myocardial contractility after endotoxic shock.
目的：观察钙增敏剂mci - 154对内毒素休克家兔器官血流量、血气、某些血生化指标及动物存活率的影响。
Objective: to investigate the effects of MCI 154, a calcium sensitizer, on organ blood flows, arterial blood gases, some plasma biochemical parameters and survival rate in endotoxic shock rabbits.
本文通过比较去甲肾上腺素和垂体后叶素对内毒素休克兔模型的作用，探讨垂体后叶素在内毒素休克中的抗休克作用。
The present study was to discuss the effect of pituitrin on rabbits with endotoxin shock by comparing with noradrenalin.
结论PDS能够明显改善内毒素休克大鼠的低血压状态，降低NOS活性、NO含量，并对其NO的生成具有抑制作用。
PDS can improve hypotension in septic shock rats, inhibit NOS activity and reduce NO content by inhibiting its production.
结论：CCK-8能减轻LPS诱导兔离体胸主动脉反应性的异常改变，这可能是CCK-8抗内毒素休克作用的机制之一。
CONCLUSION: CCK-8 improved the vascular reactivities in the presence of LPS, which may be one of the anti-endotoxic shock mechanisms of CCK.
通过家兔内毒素休克模型重点观察了磷脂酶A2（PLA2）抑制剂磷酸氯喹和地塞米松对血中PLA2及其相关脂介质的影响。
The effects of chloroquine and dexamethasone, inhibitors to phospholipase A2(PLA2), on the activity of blood PLA2 and its related lipid mediators were observed in rabbits with endotoxic shock.
结论：筛选到的短肽lttwapa能特异性结合于内毒素休克小鼠肝脏血管内皮细胞，有可能对内毒素休克的治疗具有重要意义。
CONCLUSION: the peptide of LTTWAPA specifically binds to vascular endothelial cells of liver in septic shock mice, which may have important significance for the therapy of septic shock.
本文报告了阿胶对狗内毒素性休克的治疗作用。
This paper reports the effects of Ejiao (Colla Corii Asini) on endotoxin shocks in dogs.
目的：研究大黄对内毒素性休克大鼠炎性介质作用的机制。
Objective: to study the mechanism underlying rhubarb inflammatory mediator response in endotoxic shock.
目的研究大鼠休克复苏后小肠绒毛和血内毒素含量的改变及两者的相关性。
Objective to investigate experimentally the changes of intestinal mucosal morphology in correlation with endotoxin during shock and resuscitation after trauma.
方法首先使用埃氏大肠杆菌死菌液制备大鼠内毒素性休克模型；
Methods Endotoxic shock in rats was induced by intravenous injection of dead gramegative bacteria E.
少量的内毒素就能引起脓毒血症和败血性的休克，从而导致高血压，心血管衰弱，多器官衰竭甚至死亡。
Serious endotoxin intoxication can cause sepsis and septic shocks, leading to severe hypertension, cardiovascular collapse, multiple organ failure and death.
本实验观察到家兔出血性或内毒素性休克所致的低血压能被侧脑室内注入纳洛酮，酚妥拉明或肉桂硫胺逆转。
It has been observed in rabbits that hypotension caused by haemorrhagic orendotoxic shock could be reversed by injecting naloxone, phentolamine or cinanserineinto the lateral cerebroventricle.
探讨热休克反应（HSR）对内毒素（LPS）所致MAPK信号通路的影响。
ObjectiveTo explore the effect of heat shock response(HSR) on the LPS-stimulated MAPK pathways.
用大肠杆菌内毒素（LPS）复制大鼠内毒素性休克模型，氨基胍治疗组采用氨基胍治疗。
Rats were challenged by E. coli LPS to set up the model of endotoxic shock, AG group were treated by aminoguanidine.
由于细菌毒素的作用，尤其是内毒素，败血症性休克的促炎症性反应强于其他休克。
Septic shock may be more proinflammatory than other forms because of the actions of bacterial toxins, especially endotoxin.
结论：丙酮酸乙酯可以对抗内毒素诱导的脓毒症休克犬的急性肺损伤。
CONCLUSION: Ethyl pyruvate has protective effects on acute lung injury in dogs with septic shock.
方法 我们检测了20例危重儿，18例非危重儿，10例内毒素性休克大鼠与正常大鼠的血浆内皮素与肿瘤坏死因子的含量。
Methods The concentration of serum of ET and TNF of 20 critically ill patients, 18 non-critically ill patients, 10 rats with endotoxic shock and 10 normal rats were tested.
方法 我们检测了20例危重儿，18例非危重儿，10例内毒素性休克大鼠与正常大鼠的血浆内皮素与肿瘤坏死因子的含量。
Methods The concentration of serum of ET and TNF of 20 critically ill patients, 18 non-critically ill patients, 10 rats with endotoxic shock and 10 normal rats were tested.
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