雌激素能拮抗兴奋性氨基酸毒性，稳定细胞内钙离子溶度。
Estrogen can resist the excitability amino acid toxicity and stable the solubility of calcium ion in cells.
目的：了解兴奋性氨基酸在点燃癫痫模型中的改变及其作用。
Objective to investigate the changes and action of excitory amino acids (EAA) in kindling models.
结果：视网膜缺血时兴奋性氨基酸过度释放导致神经元死亡。
Results: The release of excess excitatory amino acids caused neuronal death in ischemic retina.
目的研究兴奋性氨基酸受体激动剂对雌性大鼠青春期启动的影响。
Objective to observe the effect of excitatory amino acids receptor agonist on the onset of puberty in female rats.
缺血脑损伤大鼠皮层海马脑区的兴奋性氨基酸明显高于正常对照组。
The levels of exciting amino acids in hippocampi and cortex of ischemic brain injury rats were significant higher than those of controls.
酒精戒断综合征与海马DG区细胞外液兴奋性氨基酸神经递质含量增加有关。
The results of this study demonstrate that excitatory amino acid levels in hippocampus DG increased with subsequent ethanol withdrawal periods.
目的研究轻度高温、亚低温对局灶脑缺血组织兴奋性氨基酸（EAA）的影响。
Objective To study the effects of mild hyperthermia and mild hypothermia on excitatory amino acid(EAA) respectively.
兴奋性氨基酸转运体(EAAT)位于突触前膜、突触囊泡和神经胶质细胞膜上。
Excitatory amino acid transporters (EAAT) localizes on the presynaptic membrane, synaptic vesicle and glial membrane.
目的：探讨兴奋性氨基酸受体非竞争性拮抗剂右甲吗喃对创作性脑水肿的防治作用。
Purpose:To study the effects of excitatory amino acids receptor noncompetitive antagonist-dextromethorphan(DM)on the traumatic brain edema.
最近研究表明，自由基、兴奋性氨基酸和膜对钙的通透性是缺血性脑损伤的重要因素。
Recent studies have demonstrated that free radicals, excitatory amino acids, and membrane permeability to calcium are all important factors in ischemic brain injury.
目前，关于拟除虫菊酯对中枢神经系统兴奋性氨基酸的影响尚缺乏深入而系统的研究。
Up to now, there is no a systematic and deepgoing study about the effect of Py on EAA in CNS.
目的研究不同偶联模式的兴奋性氨基酸受体所介导非洲爪蟾卵母细胞跨膜电流的相互影响。
Aim to study the mutual influence of membrane current induced by receptors with diverse coupling modes in Xenopus oocytes.
目的研究雌激素对雄性大鼠脑缺血再灌注损伤脑组织兴奋性氨基酸(EAA)含量的影响。
Objective To study the effects of estrogen on the contents of EAA in cortex of male rats during cerebral ischemia and reperfusion.
目的探讨兴奋性氨基酸受体拮抗剂磷酸甲基谷氨酰氨酸（GAPA）对胆红素神经毒性的影响。
Objective To explore the effect of NMDA receptor antagonist glutamyl amino methyl phosphonic acid (GAPA) on the bilirubin neurotoxicity.
结论在大鼠切口疼痛模型中，鞘内注射新斯的明的镇痛作用可能与脊髓兴奋性氨基酸含量下降有关。
Conclusion the decline in the increased excitatory amino-acid contents in spinal cord induced by incisional pain is involved in the mechanisms of analgesia provided by it neostigmine.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
结论复方丹参片降低兴奋性氨基酸含量，以对抗兴奋性氨基酸的毒性，这可能是其防治ad的机制之一。
Conclusion CDST could lower the toxicity of excitatory amino acid by decreasing its contents, which may be one of the mechanisms of CDST in prevention and treatment to AD.
结论黄芩苷对大鼠脑片缺氧缺糖性损伤具有一定的保护作用，作用机制可能与其抑制了兴奋性氨基酸毒性有关。
Conclusion Baicalin possesses protective effect on OGD injury in rat brain slices, which is related to its inhibitory effect on excitatory amino acids toxicity.
目的：观察兴奋性氨基酸(EAA)作用于小鼠海马后一氧化氮(NO)含量的变化以及尼莫地平的保护作用。
Objective: to study the detrimental effect of excitatory amino acid (EAA) on the content of nitric oxide (no) in mice hippocampus, and the protective effect of nimodipine.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
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