现已知fsh受体的激活可以促进血管内皮生长因子(VEGF)的信号传导通路，而VEGF可以促进包括肿瘤组织在内的血管的生长。
Activation of the FSH receptor is known to contribute to the signaling of vascular endothelial growth factor (VEGF), a protein that stimulates the growth of blood vessels, including those in tumors.
血管内皮生长因子- C (VEGF - C)和血管内皮生长因子d (VEGF - D)可以诱导肿瘤新生淋巴管形成，并促进肿瘤淋巴道转移。
Vascular endothelial growth factors such as VEGF-C and VEGF-D can induce the growth of tumor lymphatic vessels and promote tumor metastasis through the lymphatic system.
血管内皮生长因子（VEGF）是一类能促进血管生成的细胞因子，能诱导细胞的有丝分裂和调节内皮细胞的通透性。
The vascular endothelial growth factor (VEGF) is a kind of potent angiogenic cytokine that induces mitosis and regulates the permeability of endothelial cells.
目的研究血管内皮生长因子C（VEGF C）对于淋巴管内皮细胞增殖和迁移的作用，探讨VEGF C促进淋巴管新生的机制。
Objective To investigate effects of VEGF C on proliferation and migration of lymphatic endothelial cells and explore mechanism of lymphangiogenesis induced by VEGF C.
血管内皮生长因子是促进血管生成的重要调节因子。
Vascular endothelial growth factor VEGF plays a central role in tumor angiogenesis.
近年来，有关血管内皮细胞生长因子(VEGF)在促进创口愈合和皮瓣成活方面的研究备受瞩目。
Recently, Studys related to vascular endothelial cell growth factor (VEGF) VEGF about its effect in accelerating wound healing and improving the success rate of flap are attracting more attention.
血管内皮生长因子是促进血管生成的重要调节因子。
Vascular endothelial growth factor (VEGF) plays a central role in tumor angiogenesis.
血管内皮生长因子和色素上皮细胞衍生因子是目前发现的作用最强的角膜新生血管形成促进因子和抑制因子。
Vascular endothelial growth factor and pigment epithelium-derived factor are most enhancing factor and strongest inhibitory factor of corneal neovascularization(CNV)at present.
结论：血管皮生长因子通过启动血管生成而促进肿瘤的生长，阻断血管内皮生长因子的产生可以抑制肿瘤的生长。
Conclusion: As starting angiogenesis, VEGF might promote the growth of tumor, and the inhibition of VEGF production might prevent solid tumor from growing.
新的血管生成促进了肿瘤的生长，一些多肽如血管内皮生长因子a (VEGF - A)的分泌可以刺激血管生成。
Tumor growth is fueled by the formation of new blood vessels in a process known as angiogenesis, which is stimulated by secreted peptides such as vascular endothelial growth factor a (VEGF-A).
现在他们想将VEGF(血管内皮细胞生长因子)加入到载plga支架干细胞中以观察它是否可以促进新组织中的血管生长。
They now want to try adding VEGF (vascular endothelial growth factor) to the PLGA and stem cell scaffold to see if it encourages new blood vessels to form in the new tissue.
结论：血管内皮生长因子基因转染可促进骨髓基质细胞的成骨分化。
CONCLUSION: Transfection with VEGF 165 gene can promote the osteogenic differentiation of cultured MSCs.
结论：血管内皮生长因子基因转染可促进骨髓基质细胞的成骨分化。
CONCLUSION: Transfection with VEGF 165 gene can promote the osteogenic differentiation of cultured MSCs.
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