低密度脂蛋白受体相关蛋白5 (LRP5)基因突变可影响人体骨骼。
The human skeleton is affected by mutations in low-density lipoprotein receptor-related protein 5 (LRP5).
因为低密度脂蛋白受体打扫胆固醇从血液，目标是要保持他们的人数高。
Because LDL receptors sweep cholesterol from the blood, the goal is to keep their Numbers high.
pcsk9结合低密度脂蛋白受体表面上的肝细胞和原因，这些受体打破。
PCSK9 binds to LDL receptors on the surface of liver cells and causes those receptors to break down.
“您想要更多的低密度脂蛋白受体，使阻塞pcsk9是一件好事，说：”霍顿。
"You want more LDL receptors, so blocking PCSK9 is a good thing," says Horton.
目的研究氧化低密度脂蛋白对人脐静脉内皮细胞表达CD 40中血凝素样氧化型低密度脂蛋白受体1的作用。
Objective To investigate the action of LOX-1 in Oxidized Low-Density Lipoprotein induced the expression of CD40 in cultured human umbilical vein endothelial cells.
目的探讨中国北方绝经前妇女低密度脂蛋白受体相关蛋白5（LRP5）基因Q89R、A1330V多态性与骨密度关系。
Objective To investigate the association of lipoprotein receptor-related protein 5(LRP5) gene Q89R, A1330V polymorphism with bone mineral density(BMD) in premenopausal Northern Chinese women.
目的：观察极低密度脂蛋白受体(VLDL -R)的两种亚型在糖尿病患者脂肪组织中的分布和表达，以探讨其变化及意义。
AIM: to observe the distribution and expression of two subtypes of very low density lipoprotein receptor (VLDL r) in diabetic adipose tissues to investigate their changes and significance.
为了了解LXR如何调节低密度脂蛋白受体，作者研究了LXR靶基因的转录，并确定了一个能编码肌球蛋白互动蛋白的基因，IDOL。
To understand how LXR regulates LDLR, the authors examined transcriptional target genes of LXR and identified a gene that encodes a myosin interacting protein, IDOL.
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
介导氧化修饰低密度脂蛋白的受体种类繁多，尤其是清道夫受体a。
There are many types of receptors secreted the oxidized low density lipoprotein, especially scavenger receptor a.
LDL受体介导的低密度脂蛋白胆固醇水平的升高是致动脉性粥样硬化的主要原因，与冠心病危险性的增加密切相关。
The LDL receptor defects cause the elevation LDLcholesterol in plasma. This is the main reason for atherosclerosis and is closely related to the risk of coronary heart disease (CHD).
本文研究了小鼠腹腔巨噬细胞极低密度脂蛋白（VLDL）受体的调节。
The regulation of the VLDL receptor on mouse peritoneal macrophages was studied.
例如，家族性血胆脂醇过多症(FH)通常起源于编码低密度脂蛋白(LDL)受体的LDLR突变。
For instance, familial hypercholesterolemia (FH) typically results from mutations in LDLR encoding the low-density lipoprotein (LDL) receptor.
建立具有正常极低密度脂蛋白（VLDL）受体结合功能的小分子受体片段模型并借助此模型研究VLDL受体与配体结合的作用机制。
The purpose of the paper is to build a module of small molecular receptor fragments charactering normal VLDL-R binding function and study the mechanism of VLDL-R binding to ligand.
建立具有正常极低密度脂蛋白（VLDL）受体结合功能的小分子受体片段模型并借助此模型研究VLDL受体与配体结合的作用机制。
The purpose of the paper is to build a module of small molecular receptor fragments charactering normal VLDL-R binding function and study the mechanism of VLDL-R binding to ligand.
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