而更危险的甲基汞(可损伤神经)的半衰期是44天。
The more dangerous methyl mercury, known to damage nerves, has a half-life of 44 days.
结论：新生大鼠缺氧缺血性脑损伤神经元退行性变形式多样。
Conclusion: This neuron degeneration of HIE in neonatal rats displayed a morphological diversity.
他们说，这种药物看来能够除去通常阻止受伤神经细胞重生轴突的障碍。
Recovery from spinal cord injuries is often impossible because of the formation of scar tissue, which impedes the growth of new nerves.
结论：电针可提高损伤神经神经元内源性NGF水平，促进神经功能恢复。
CONCLUSION: Electroacupuncture therapy can increase the level of endogenous NGF, and improve the functional rehabilitation of injured nerve.
淀粉样β蛋白本身可能损伤神经细胞或者有它聚集形成的老年斑才是罪魁祸首。
Amyloid beta itself might be injuring nerve cells or the plaques, made of accumulations of amyloid beta, could be the culprits.
胡黄连对脑I /R损伤神经元有保护作用，其机制可能与上调脑内NGF表达有关。
Picrorhizae can protect the neuron from I/R injury by increasing the expression_r_r of NGF in brain.
就铅接触而言—甚至较低量—持续接触可产生严重影响，如贫血、不舒服和损伤神经系统。
In the case of lead exposure - even at relatively low levels - continuous exposure may have severe effects, such as anaemia, malaise, and damage to the nervous system.
虽然多数该类肿瘤不会像其他癌症那样扩散，但是在脊髓中多余的增生会进一步损伤神经。
Even though most such tumors do not spread like other cancers, any unwanted growth in the spinal cord can further damage nerves.
结论：靶肌肉注射甲钴胺可促进周围神经的再生，高剂量的甲钴胺可以更好地发挥其对损伤神经的营养作用。
CONCLUSION: Target muscular injection of methyl cobalamin can promote the regeneration of peripheral nerve, and methyl cobalamin of high dosage can better play its role in nourishing injured nerve.
结论tPA和PAI1参与周围神经挤压伤后的病理生理改变，在损伤神经修复过程中具有一定的作用。
Conclusions t pa and pai 1 not only participate in the pathophysiological changes of peripheral nerves after crush injury, but have some effects on peripheral nerve repair as well.
结论：周围神经损伤后早期应用微波辐射治疗能增加GAP - 43的表达，促进损伤神经再生及功能恢复。
Conclusion: microwave could enhance the expression of GAP-43 and promote the regeneration and functional recovery of the injured peripheral nerve after early microwave irradiation.
经损伤坐骨神经的SD大鼠服用后的实验结果表明，“神经生长液”口服液合剂，具有促进损伤神经恢复功能的作用。
The results of the experiment of lesioned sciatic nerve with the treatment of the Nerve Growth Liquid in SD rat indicated that the drug may promote the function recovery of lesioned nerve.
结论香椿果抗补体活性多酚XCG-7能促进SH-SY5Y细胞的生长，对补体损伤神经细胞具有一定的保护作用。
Conclusion The anticomplementary polyphenol XCG-7 from the seed of Toona sinensis can promote SH-SY5Y cell growth, and protect SH-SY5Y cells from complement-induced injury.
结论STAT 3蛋白表达增强可能介导缺血性脑损伤的信号转导过程，其在脑缺血损伤神经元凋亡过程中起关键作用。
Conclusion the expressions of STAT3 increased might indicate the transmission course in ischemic nerve cell signal, and play a key role in neuron apoptosis of ischemic brain injury.
目的：研究神经生长因子(NGF)对兔局灶性脑缺血再灌注损伤神经功能修复的影响，并利用MR成像技术进行评价。
Objective: To investigate the influence of NGF on neural functional recovery in focal cerebral ischemia reperfusion injury in rabbits and evaluate with MR imaging.
因为这种治疗方法会损伤神经，对舌头造成永久或暂时麻痹，有很高的风险，所以口腔外科中不再将此法作为标准治疗方法。
There is a high risk of permanent or temporary numbness of the tongue due to damage of the nerve with this treatment and it is no longer recommended as a standard treatment in oral surgery.
应用免疫细胞化学技术研究BDNF、GDNF蛋白在体外培养正常神经干细胞和缺氧损伤神经干细胞中的表达以及脑溢安的影响。
Using immunocytochemistry to study the expression of BDNF and GDNF in normal NSC and NSC that had been damaged by hypoxia.
应用免疫细胞化学技术研究BDNF、GDNF蛋白在体外培养正常神经干细胞和缺氧损伤神经干细胞中的表达以及脑溢安的影响。
Using immunocytochemistry to study the expression of BDNF and GDNF in normal NSC and NSC that had been damaged by hypoxia.
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