从新陈代谢的角度讲，它们仅仅是处于一种休眠状态，等抗生素的威胁过后重新开始活跃，这也是受感染的病人久久无法痊愈的原因。
They just power down, metabolically speaking, and then wait until the coast is clear (of antibiotics) to come back to life. Which is why the poor patient just can't seem to clear the infection.
“二甲胺四环素降低了病毒脱离休眠状态的感染T细胞的能力，”Szeto解释说。
"Minocycline reduces the capability of the virus to emerge from resting infected t cells," Szeto explains.
种子休眠机理主要围绕透性、抑制剂作用和光敏素转化等方面的研究而建立。
Seed dormancy mechanisms mainly focus on seed permeability, inhibitor action and photofrin transformation.
种子休眠机理主要围绕透性、抑制剂作用和光敏素转化等方面的研究而建立。
Seed dormancy mechanisms mainly focus on seed permeability, inhibitor action and photofrin transformation.
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