这种代偿性肥大随着时间的推移逐渐失去代偿，随着心肌细胞消失、纤维化，心室功能障碍。
This compensatory hypertrophy is pathologic because the process is maladaptive over time, with eventual myocyte loss, fibrosis, and ventricular dysfunction.
这种代偿性肥大随着时间的推移逐渐失去代偿，随着心肌细胞消失、纤维化，心室功能障碍。
This compensatory hypertrophy is pathologic because the process is maladaptive over time, with eventual myocyte loss, fibrosis, and ventricular dysfunction.
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