目的：研究高糖对人肾小球系膜细胞丝裂原蛋白激酶（MAPK）活性的影响，探讨其在糖尿病肾病发病中的作用。
Objective To study the effects of high glucose on activity of mitogen activated protein kinase (MAPK) in cultured human mesangial cells and to explain the mechanism of diabetic nephropathy.
氨基核苷嘌呤霉素诱导肾病变是人原发性肾病综合症的模型，抑制培养的肾小球上皮细胞整联蛋白的表达。
Puromycin aminonucleoside-induced nephrosis is a well-described model of human idiopathic nephrotic syndrome, suppressing expression of integrin expression in cultured glomerular epithelial cells.
方法采用放射免疫分析法测定了32例无糖尿病肾病和5 2例糖尿病肾病患者血清中肌红蛋白含量，并与35名正常健康人作对照。
Methods Level of Mb in serum was measured in 32 cases of non diabetic nephrosis and 52 cases of diabetic nephrosis and compared with 35 normal controls.
方法采用放射免疫分析法测定了32例无糖尿病肾病和5 2例糖尿病肾病患者血清中肌红蛋白含量，并与35名正常健康人作对照。
Methods Level of Mb in serum was measured in 32 cases of non diabetic nephrosis and 52 cases of diabetic nephrosis and compared with 35 normal controls.
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