上皮细胞培养基是专门为正常人类上皮细胞体外培养设计的最适于其生长的培养基。
Epithelial cell medium (EpiCM) is a complete medium designed for optimal growth of normal human epithelial cells in vitro.
该培养体系为深入研究人类支气管上皮细胞多阶段癌变的细胞和分子机制提供了一种理想的生物材料。
This culture system provides a potential tool for the study of cell and molecular mechanism in the multistage carcinogenesis of human bronchial epithelial cells.
目的研究塞替派对人类支气管上皮细胞的致癌转化效应。
Objective To investigate the malignant transformation effects of human bronchial epithelial cells induced by thiotepa.
目的：为探讨热休克蛋白- 70对人类糖尿病性白内障晶状体上皮细胞的影响。
AIM: To study the effect of heat shock protein-70 (HSP-70) on lens epithelial cells (LECs) in human diabetic cataract.
部分胰岛细胞、胰腺外分泌细胞和所有导管上皮细胞均表达人类血型A抗原。
The human blood a type antigen was highly expressed on some islet cells, some exocrine secretory cells and all the duct epithelium.
观察了烟草特异亚硝胺nnk诱发HPV -18永生化人类支气管上皮细胞(BEP2D)恶性转化过程中细胞生物学特征的变化。
The malignant transformation of HPV 18 immortalized human bronchial epithelial cell (BEP2D) induced by tobacco specific nitrosamine NNK and its biological characteristics were investigated.
结果显示，人类晶体上皮细胞在体外生存能力有限，体外的生长能力与供体年龄呈负相关。
The results revealed that the in vitro survival capacity of the human lens epithelial cells is limited and the proliferative capacity of the cells is negatively cor-related to the donor age.
结果在扫描电镜下，人类卵巢上皮细胞可区分为扁平细胞和立方细胞。
Results the human ovary epithelium was composed of cuboidal and flat cells.
志贺菌属的细菌以人类为特异性宿主，感染人类肠上皮细胞，多导致痉挛性腹痛、腹泻、发烧等症状，是细菌性痢疾最为常见的病原菌。
Shigella spp. is human host-specific pathogens that infects intestinal epithelial cells. It is the causative agent of dysentery which is followed by cramps, diarrhea and fever.
病毒主要侵犯人类呼吸道，一方面在呼吸道上皮细胞和血管内皮细胞内复制，损伤肺组织，破坏气体交换屏障；
Virus mainly affects the human respiratory tract. It can replicate in airway epithelial cells and endothelial cells, damage lung tissue, and destroy gas exchange barrier.
病毒主要侵犯人类呼吸道，一方面在呼吸道上皮细胞和血管内皮细胞内复制，损伤肺组织，破坏气体交换屏障；
Virus mainly affects the human respiratory tract. It can replicate in airway epithelial cells and endothelial cells, damage lung tissue, and destroy gas exchange barrier.
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