结论OSAHS患者心律失常的发生与呼吸暂停低通气指数，低氧血症及交感紧张密切相关。
The occurrence of cardiac arrhythmia in patients with OSAHS is closely related with AHI and nocturnal hypoxemia and sympathetic tone.
在极度紧张的情况下，人体的交感神经系统为身体有力的求生行为做了准备工作。
Under acute stress, the body's sympathetic nervous system prepares the body for sustained, vigorous action.
交感神经高度紧张和兴奋。
急性失交感神经支配的皮肤或者肢端由于失去了交感性缩血管神经紧张作用，皮肤是温暖的。
In acutely sympathetically denervated areas, the skin or extremity is warm because of loss of sympathetic vasoconstrictor tone.
肾脏肾小球旁体的交感神经支配释放肾素；血管紧张素刺激大脑自主中枢，增加交感冲动发放。
Sympathetic innervation of the juxtaglomerular apparatus in the kidney releases renin; angiotensin stimulates autonomic centers in the brain to increase sympathetic discharge.
低血钾的产生主要由于交感-儿茶酚胺系统紧张性增高所致。
The hypopotassemia may be mainly caused by in-creased tone in sympathetic-catecholamine system.
雷诺现象的发作也可出现在交感神经系统刺激之后(如情绪紧张，突然惊吓)。
An attack of RP may also occur after stimulation of the sympathetic nervous system (such as emotional stress, sudden startling).
肾素-血管紧张素系统作用机制的第一步是肾近球细胞受到低血压、交感神经兴奋、低血容量及低钠流量的刺激而分泌肾素。
The first step in the RAS cascade is the secretion of the enzyme renin by renal juxtaglomerular cells in response to low blood pressure, sympathetic activation, hypovolemia, and low sodium flux.
高血压左心室肥厚(LVH)与肾素-血管紧张素-醛固酮系统及交感神经-儿茶酚胺系统的关系比较明确。
The relationship between hypertensive left ventricular hypertrophy (LVH) and renin-angiotensin-aldosterone system (RAAs) the sympathetic nervous-catecholamine (ca) system are clear.
高血压左心室肥厚(LVH)与肾素-血管紧张素-醛固酮系统及交感神经-儿茶酚胺系统的关系比较明确。
The relationship between hypertensive left ventricular hypertrophy (LVH) and renin-angiotensin-aldosterone system (RAAs) the sympathetic nervous-catecholamine (ca) system are clear.
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