我们假设细胞蛋白s -亚硝基化的变化会引起高血糖介导的内皮功能障碍。
We hypothesized that alterations in cellular protein S-nitrosylation may contribute to hyperglycemia-induced endothelial dysfunction.
其中之一就是S-亚硝基化，我们已经和霍普金斯的一个研究组合作了一些研究，结果一种蛋白，谷氨酰胺转移酶2，似乎能被这一S-亚硝基化过程所影响。
One area is S-nitrosylation, where we've been doing some work with a group at Johns Hopkins, the protein, transglutaminase2, seems to be effected by this process S-nitrosylation.
亚硝基脲的抗癌与致癌机理是非常接近的，都能够使DNA碱基发生烷化，进一步形成DNA股间交联。
The anticancer mechanism of nitrosoureas is very similar to the carcinogenic mechanism, both of them can alkylate DNA bases and form DNA cross-linking further.
亚硝基脲的抗癌与致癌机理是非常接近的，都能够使DNA碱基发生烷化，进一步形成DNA股间交联。
The anticancer mechanism of nitrosoureas is very similar to the carcinogenic mechanism, both of them can alkylate DNA bases and form DNA cross-linking further.
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