目的探讨角膜基质细胞组织因子途径抑制物2 (TFPI - 2)的表达情况，为角膜新生血管的基因治疗提供方法。
Objective to observe the expression of TFPI-2 in corneal keratocytes in vitro and investigate the experimental basis for curing corneal neovascularization with gene-therapy.
如图所示为严重化学伤后早期发生的角膜新生血管形成。
The image shown demonstrates the effects of a severe chemical injury with early corneal neovascularization.
以SD角膜新生血管化大鼠为受体，建立大鼠高危穿透性角膜移植动物模型。
SD rats with corneal vascularization were taken as recipients to establish animal model of high-risk penetrative corneal transplantation.
移植治疗无菌性角膜溃疡术后，1~2周有新生血管侵入组织工程角膜基质植片边缘，植片为灰白色半透明状；
From the 1st week to the 2nd week after transplantation, the implant's margin of tissue engineering cornea stromas were invasioned by new vessels and became grey and semitransparent;
移植治疗无菌性角膜溃疡术后，1~2周有新生血管侵入组织工程角膜基质植片边缘，植片为灰白色半透明状；
From the 1st week to the 2nd week after transplantation, the implant's margin of tissue engineering cornea stromas were invasioned by new vessels and became grey and semitransparent;
应用推荐