脂质过氧化和低密度脂蛋白的氧化修饰在动脉硬化的发生中起着极其重要的作用。
There is increasing evidence that lipid peroxidation and oxidative modification of low density lipoprotein is important in atherogenesis.
这项工作显示了氧化低密度脂蛋白在骨桥蛋白基因表达中的作用，并且进一步解释了氧化应激在细胞因子调节中的作用。
This work demonstrates the role of OxLDL in the expression of the OPN gene and further highlights the role of oxidative stress in the regulation of this cytokine.
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
结论：循环氧化低密度脂蛋白水平的升高与主动脉瓣膜狭窄中的瓣膜组织纤维钙化重构有密切联系。
Conclusion: Increased level of circulating ox-LDL is associated with worse fibrocalcific remodelling of valvular tissue in AS.
目的研究氧化低密度脂蛋白对人脐静脉内皮细胞表达CD 40中血凝素样氧化型低密度脂蛋白受体1的作用。
Objective To investigate the action of LOX-1 in Oxidized Low-Density Lipoprotein induced the expression of CD40 in cultured human umbilical vein endothelial cells.
并测定标准血浆脂蛋白，低密度脂蛋白颗粒大小以及血浆中循环氧化低密度脂蛋白（4E6抗体）水平。
The standard plasma lipid profile, the size of LDL particles and the plasma level of circulating ox-LDL (4E6 antibody) were determined.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
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