我们推测在剧烈运动中炎症反应可能会抑制内皮祖细胞从骨髓中释放。
We speculate that under intensive exercise the inflammatory situation may suppress liberalisation of progenitor cells from the bone marrow.
目的探讨环氧化酶（COX）1、2在缺血心肌血管生成和内皮祖细胞动员中的作用及机制。
Objective To investigate the effect of COX1 and COX2 on angiogenesis and endothelial progenitor cell mobilization in rats with experimental myocardial infarction(MI).
本研究探讨骨髓内皮细胞分泌的造血抑制因子在扩增造血祖细胞中的作用并初步探讨其作用机制。
In this study, the roles of hematopoietic inhibitors elaborated by bone marrow endothelial cells in the proliferation and differentiation of hematopoietic progenitors were investigated.
流式细胞分析显示神经生长因子处理后能够增加C57BL/6小鼠外周循环系统中的内皮祖细胞数量急剧增加。
Flow cytometric analysis revealed that NGF treatment increased the number of EPCs in the peripheral circulation of C57BL/6 mice.
流式细胞分析显示神经生长因子处理后能够增加C57BL/6小鼠外周循环系统中的内皮祖细胞数量急剧增加。
Flow cytometric analysis revealed that NGF treatment increased the number of EPCs in the peripheral circulation of C57BL/6 mice.
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