当血管壁受损，胶原蛋白和组织因子暴露，循环血小板通过血管性血友病因子的作用发生黏着，并伴形状改变和ADP的释放。
When vessel walls are damaged, collagen and tissue factor are exposed, and circulating platelets adhere via von Willebrand factor and undergo a change in shape with the accompanying release of ADP.
微栓塞能够引起组织缺血，红细胞片断引发溶血性贫血，或者血小板和凝血因子消耗导致出血。
Microscopic thrombi can produce tissue ischemia, red cell fragmentation leading to a hemolytic anemia, or hemorrhage due to consumption of platelets and clotting factors.
但是有一个可能是血液替代品里面的血红蛋白分子夺取氧化一氮分子，这些分子一般发挥舒张血管和组织血小板凝集作用。
But one possibility is that hemoglobin molecules in substitute blood capture nitric oxide molecules that normally dilate blood vessels and keep platelets from getting sticky.
损伤之后，血小板通过被称为黏附的过程黏着于含胶原的皮下组织形成血管内皮内衬。
Following injury to the endothelial lining of blood vessels, platelets adhere to collagen-containing subendothelial tissue by a process called adhesion.
体外培养1周后，取组织工程瓣膜及未种细胞的空支架做血小板黏附试验。
After cultured for 1 week, the tissue engineered heart valve and unseeded scaffold were tested by platelet adhesion experiments.
通过大体和扫描电镜两种方法观察富含血小板血浆凝胶的结构，探索其用于组织工程载体的可能性。
To observe the structure of platelet-rich plasma gel by gross observation and scanning electron microcopy so as to estimate the possibility of it used as a scaffold of tissue engineering.
目的：探讨富血小板血浆对大鼠牙周组织再生的作用。
AIM: To investigate the effect of platelet-rich plasma (PRP) on the regeneration of rats' periodontal tissue.
结论：再生脾组织T淋巴细胞亚群分布发生改变，对防止自身血小板抗体的产生有益。
Conclusion: the distribution of T-lymphocyte changes in regenerated spleen and is benefit to inhibit the PAIgG.
与红细胞及血小板不同，白细胞能够脱离血液，并且游走到组织内，以完成它们摄取、吞噬细菌以及真菌的使命。
Unlike red cells and platelets, the white cells are capable of leaving the blood and moving into the tissues where they can ingest, or eat, bacteria or fungi.
动脉血栓通常形成于血液湍流区和粥样癍块破裂区，这些部位的内皮下组织暴露于血小板和凝血蛋白。
Arterial thrombi usually form in regions of disturbed flow and at sites of rupture of an atherosclerotic plaque, which exposes the thrombogenic subendothelium to platelets and coagulation proteins;
本文对十例危重风湿病出血的病人，进行了血小板功能、凝血象、组织病理检查，确诊出血原因为血管炎引起。
The article reported the function of platelet, blood coagulation and pathologic examination of 10 cases with rheumatism who complicated hemorrhage.
本文对十例危重风湿病出血的病人，进行了血小板功能、凝血象、组织病理检查，确诊出血原因为血管炎引起。
The article reported the function of platelet, blood coagulation and pathologic examination of 10 cases with rheumatism who complicated hemorrhage.
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