迪斯凯尔想知道，为什么心肌细胞不取代疤痕组织呢？
Why, Discher wondered, don't heart muscle cells then replace the scar tissue?
他成功地对老鼠的结缔组织细胞进行了基因改造，使它们发育成为心肌细胞，并已经在患有心脏病的老鼠的心脏上对这些细胞进行了测试。
He has managed to reprogram mouse connective-tissue cells so that they develop into cardiac muscle cells, and has tested them in the hearts of mice that have had heart attacks.
这种方法临时以纤维蛋白凝胶来培养大鼠的心肌细胞，当细胞形成组织后去除纤维蛋白凝胶。
The approach USES a fibrin gel to support rat cardiac cells temporarily, before the fibrin breaks down as the cells organize into tissue.
目的观察中药复方温心汤对家兔心肌缺血再灌注损伤心肌细胞凋亡以及病理组织学改变的影响。
Objective To observe mainly the effect of the Chinese traditional medicine (compound Wenxin soup) on myocyte apoptosis and path-histology change in rabbits ischemia referpusion injury.
干细胞具有分化成多种机体组织细胞，包括心肌细胞的潜能。
Stem cells have the ability to form many fully differentiated cells of the body, including cardiac myocytes.
测定心肌病毒滴度、心肌细胞凋亡率和坏死率，计算心肌病理组织学积分。
The virus titer in myocardium, percentage of apoptosis and necrosis of myocytes were detected, myocardial histopathologic scores were counted.
心肌细胞超微结构观察结果：糖尿病组心肌组织内可见凋亡的心肌细胞。
Results of ultrastructure of cardiac myocyte: Apoptosis could be observed in myocardial cell in diabetic group.
他们通过将心肌细胞置于周期性扭转的机械装置中，模拟在体环境，成功地在体外再造了心肌组织。
They successfully regenerated myocardial tissue in vitro, through placing myocardial cells in a periodically-twisting mechanical device, an in vivo simulating environment.
对照组心脏标本中可见瘢痕组织；心肌细胞结蛋白染色阴性，肌钙蛋白i染色阳性。
Scar tissue was seen in the control group, and desmin was negative and Troponin I was positive in cardiomyocytes.
结果在心衰大鼠的左、右心室心肌组织中存在有凋亡的心肌细胞，而在对照组大鼠心肌组织中未见到心肌细胞调亡。
Results Apoptosis was detected in the myocardium of failing hearts of rats. No apoptosis was found in myocardium of rats in control group.
目的：观察低硒喂养对大鼠心肌组织脂质过氧化物、心肌细胞超微结构及细胞凋亡的影响。
AIM: to investigate the effects of low-selenium diet on rats 'myocardial lipid peroxidize and apoptosis.
结果(1)he法：uc组大鼠心肌组织结构正常，IH组心肌灶性变性，心肌细胞肿胀，间质中有炎性细胞浸润。
Results (1) he method: the changes of vessel in heart, lung and kidney in UC group were not found, while the rat in IH group were found for myocardial cell swelling and inflammatory cell infiltration.
方法制备心肌组织缺氧损伤模型，观察不同剂量的地高辛抗血清对缺氧损伤心肌组织内源性洋地黄样因子水平和心肌细胞膜atp酶活性的影响。
Methods it was observed that different concentration anti_digoxin antiserum effect on endogenous digitalis_like factor and cell membrane ATPase activity in hypoxic myocardium model.
各种心脏疾病发生后，因为心肌细胞增殖能力极低，坏死心肌有纤维组织替代，最终导致心力衰竭。
The necrotic myocardium after various kinds of heart diseases often is replaced by fibrous tissue because of its the very low regenerative potentiality, which will then result in heart failure.
结论T型钙离子通道对于心肌细胞以及心脏传导组织的形成起到了一定的作用。
Conclusion T-type calcium channels play an important role in the morphogenesis of myocardium and cardiac conduction system during mouse embryonic heart development.
目的：从人胎盘组织中提取干细胞，并诱导其向心肌细胞分化。
AIM: to isolate the stem cells from human placenta and induce them into cardiomyocytes.
研究人员希望通过新组织的移植进而修复受损的心肌，因此，心肌细胞是再生医疗中最紧缺的细胞之一。
Heart cells are one of the most sought-after cells in regenerative medicine because researchers anticipate that they may help to repair injured hearts by replacing lost tissue.
结论：二氢槲皮素能有效抑制ISO所致心肌组织缺血，保护心肌细胞免受损伤，从而起到治疗冠心病的作用。
Conclusion: Dihydroquercetin had an effective role to treat coronary heart disease by inhibiting myocardial tissue ischemia induced by ISO and protecting myocardial cells from injury.
结论：GH可使心肌细胞、骨骼肌细胞肥大而不是间质组织增生。
Conclusion GH can induce hypertrophy of cardiac muscle cells and skeletal muscle cells but not interstitial proliferation.
方法： 制备心肌组织缺氧模型，观察不同剂量的地高辛抗血清对缺氧损伤心肌组织内洋地黄素水平和心肌细胞膜ATP酶活性的影响。
METHODS: It was observed that different concentration of anti-digoxin antiserum effect on endoxin and cell membrane ATPase activity in hypoxic myocardium model.
方法： 制备心肌组织缺氧模型，观察不同剂量的地高辛抗血清对缺氧损伤心肌组织内洋地黄素水平和心肌细胞膜ATP酶活性的影响。
METHODS: It was observed that different concentration of anti-digoxin antiserum effect on endoxin and cell membrane ATPase activity in hypoxic myocardium model.
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