The flavonoids inhibited the secretion of various inflammatory cells called cytokines in H5N1-infected cells, but had no effect on noninfected cells or tumor necrosis factor, a key inflammatory protein.

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In mice, a protein connected to inflammatory reactions appeared to be switched on when the animals were given a high fat, high sugar diet.

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This protein spurs the body to produce crucial inflammatory molecules called leukotrienes, which had been thought to be involved mainly in asthma.

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The model also suggested that an anti-inflammatory drug called celecoxib should bind with CDH11, a protein which plays an important role in the development of both rheumatoid arthritis and metastatic breast cancer.

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This binding process stimulates production of a protein called interleukin-10 (IL-10) that acts as an anti-inflammatory compound and which helps keep the immune system's response to the infection in check.

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