A key aspect of Alzheimer's is sticky clumps of an abnormal protein in the brain called beta amyloid plaques.
阿尔兹·海默氏症主要是由于脑部聚集结块的异常蛋白质，名为be ta淀粉状蛋白斑。
Amyloid is an abnormal protein usually produced by cells in your bone marrow that can be deposited in any tissue or organ.
淀粉样蛋白通常由骨髓细胞合成，可以在各个组织和器官中沉积。
Until now, it was not clear why the disease only affects nerve cells and how the abnormal protein agglutination is triggered.
直到现在位置，人们不清楚为什么这种病只涉及到神经细胞、正常蛋白凝集又是如何触发的。
Conclusion (1) Li ver fibrosis is accompanied with abnormal protein synthesis and asition, a s well as the enhanced activity of glycolysis;
结论在肝纤维化过程中伴有蛋白质合成与分解异常和糖酵解活动增强；
With the molecular progress of biological research, the abnormal protein molecules related to various neurodegeneration diseases have been identified.
随着生物学研究向分子层次深入，人们已经确知了与各种神经退行性疾病相关的异常蛋白质分子。
It has been proved that ERS can interfere the differentiation of chondrocyte, decrease the synthesis of abnormal protein, attenuate the injury of cell.
有研究证实内质网应激能影响软骨细胞的分化，同时也抑制了软骨细胞的合成功能，减少异常蛋白的合成，减轻应激对细胞的损害。
Mistakes in a cell's reading of RNA into protein can lead to the production of an abnormal protein, and this can result in abnormal cell function or death.
细胞利用这种监视机制来找到并破坏异常rna。RNA编译成蛋白时的错误会产生异常的蛋白，这最终导致细胞功能异常甚至死亡。
This invention relates to methods and compositions for the treatment or alleviation of Alzheimer's disease and of other conditions related to abnormal protein aggregation.
本发明涉及用于治疗或减轻阿尔茨海默氏病和其它与异常蛋白聚集有关的病症的方法和组合物。
What we are doing here is precisely matching a treatment to a specific type of cancer cell mutation and abnormal protein signaling pathways that may activate cancer cell growth.
我们在此所做的就是精确匹配治疗方案。一种治疗方案针对一种特殊类型的癌症突变细胞和可能激活癌症细胞生长的异常蛋白信号通路。
These abnormal LPL cells produce large amounts of the protein immunoglobulin and macroglobulin.
这些异常的LPL细胞产生大量的蛋白免疫球蛋白和巨球蛋白。
Gluten intolerance is caused by an abnormal immune response to gluten, the major protein in wheat and some other grains.
麸质是一种蛋白质，它是小麦和其它谷物的主要成分，对麸质不正常的免疫反应则是麸质不耐症的最终病因。
With a cow's-milk allergy, the immune system has an abnormal reaction to milk protein.
而对牛奶过敏是指免疫系统对牛奶蛋白质有异常反应。
The two distinct diseases are marked by an abnormal accumulation of the same protein - a startling, two-for-one discovery described in the journal Science.
这两种截然不同的疾病被发现有相同蛋白的异常聚集-这个令人吃惊的、二合一的发现发表在《科学》杂志上。
The nutritional status was well compared with the abnormal ratio of hemoglobin and serum protein, and A/G reverse was increased in some middle malnutrition patients.
营养状况与血红蛋白及血清蛋白异常率呈正相关，中度营养不良重病人的白球比值倒置比例明显增高。
There exist hypermethylation, abnormal transcription of FHIT gene and down-regulation expression of FHIT protein in liver cancer.
肝癌中存在FHIT基因高甲基化、转录异常、FHIT蛋白表达下降。
Because the island of langerhans rope relative or insufficient causes absolutely hefts, the fat and the protein metabolism is abnormal.
由于胰岛索相对或绝对不足所引起的掂、脂肪和蛋白质代谢失常。
The majority of BTCC in this group showed abnormal expression of multiple items of products of gene protein.
本组BTCC大多数表现为多项基因蛋白产物表达异常。
Objective: to explore the risk factors of P16 protein abnormal expression in colorectal cancer (CRC).
目的：探讨大肠癌p 16基因异常表达的危险因素。
Objective: to study the abnormal crystalline protein X36 in cataract SD rat and to explore the relationship be-tween X36 and cataractogenesis.
目的：研究半乳糖诱导白内障大鼠异常晶状体蛋白x 36的出现及其与白内障形成的关系。
These findings suggest a role for ANKHD1 as a scaffolding protein that may be associated with the abnormal phenotype of leukemia cells.
这些发现显示的作用ANKHD1作为脚手架蛋白可能与异常的表型白血病细胞。
Hemoglobin s is an abnormal version of the protein hemoglobin.
血红蛋白是一种非正常血红蛋白。
They revealed that in normal cells, BCL9L helps activate a protein called caspase-2 in response to an abnormal number of chromosomes, triggering a self-destruct sequence.
他们发现在正常的细胞中，BCL9L帮助激活一种叫做半胱天冬酶-2的蛋白质以应对异常数量的染色体，引发自毁程序。
Objective To determine the sensitivity and false-positive rate of maternal serum pregnancy associated plasma protein a (PAPP-A) screening for abnormal fetal karyotype.
目的分析妊娠相关血浆蛋白a (PAPP - A)在产前筛查胎儿染色体病时的灵敏度和假阳性。
Aim: to explore the expression and abnormal activation of mitogen-activated protein kinase (MAPK) in the development and progression of cerebral gliomas.
目的：研究人脑胶质瘤组织中丝裂原激活蛋白激酶(MAPK)的表达及活化情况。
Aim: to explore the expression and abnormal activation of mitogen-activated protein kinase (MAPK) in the development and progression of cerebral gliomas.
目的：研究人脑胶质瘤组织中丝裂原激活蛋白激酶(MAPK)的表达及活化情况。
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