甘精胰岛素不能治疗餐后高血糖。
餐后高血糖是卒中的危险因素吗？
目的：探讨餐后高血糖与心血管疾病发生的相关性。
Objective: To investigate the postprandial hyperglycemia and cardiovascular diseases related to each other.
氧化损伤对餐后高血糖诱导的内皮功能障碍起到重要作用。
Oxidative injury might play an important role in the endothelial dysfunction induced by postprandial hyperglycemia.
结论阿卡波糖是降低餐后高血糖的一种有效而安全的降糖药。
Conclusions Acarbose was an effective and safe drug in lowering postprandial hyperglycemia.
结论年龄、餐后高血糖、血压与老年人大血管损害密切相关。
Conclusion Age, postprandial hyperglycemia and hypertension are closely related to lesions of big vessels in the elderly.
目的探讨2型糖尿病患者餐后高血糖状态与血管并发症的关系。
Objective to probe into the relationship of postprandial glucose and vascular disease in type 2 DM.
根据以上机制，我们认为餐前服用些许蛋白质可以降低2型糖尿病患者的餐后高血糖。
We hypothesized that a protein preload would reduce the postprandial glycemic excursion in type 2 diabetic patients by these mechanisms.
该研究结果并不支持降低餐后高血糖对预防或减少糖尿病心血管疾病有特殊作用的观点。
The results from the NAVIGATOR study do not support the contention that reducing postprandial hyperglycemia has a specific role in preventing diabetes or reducing cardiovascular disease.
结果就是发生高血糖特别是餐后高血糖，这是因为进餐后葡萄糖进入循环但不能被骨骼肌吸收。
The result of this is to have hyperglycaemia, particularly in the post-prandial state, so when a meal is consumed, the glucose gets into the circulation and is not absorbed by the skeletal muscle.
多年来，广大的医务人员和患者比较重视对空腹血糖的控制，而对餐后高血糖的危害性缺乏正确认识。
In recent years, Medical members and patients are all pay more attention to the controlling of blood-fasting sugar, while they are lack of recognition for danger of postprandial hyperglycemia.
目的：评价餐前进食乳清是否可以延缓胃排空、促进肠激素的分泌以及降低2型糖尿病患者的餐后高血糖。
OBJECTIVE We evaluated whether a whey preload could slow gastric emptying, stimulate incretin hormones, and attenuate postprandial glycemia in type 2 diabetes.
第一相胰岛素反应丧失的后果为肝脏未能迅速胰岛素化，延迟了对肝葡萄糖输出的抑制，从而引起餐后高血糖。
Loss of first-phase response has metabolic consequences: lack of rapid insulinization of the liver delays the suppression of hepatic glucose output and causes therefore postprandial hyperglycemia.
第一相胰岛素反应丧失的后果为肝脏未能迅速胰岛素化，延迟了对肝葡萄糖输出的抑制，从而引起餐后高血糖。
Loss of first-phase response has metabolic consequences: lack of rapid insulinization of the liver delays the suppression of hepatic glucose output and causes therefore postprandial hyperglycemia.
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