目的：评价酶免疫分析法（ELISA）体外检测过敏原在荨麻疹病因筛查中的临床应用价值。
Objective: To evaluate the clinical value of in vitro allergen detection by ELISA in aetiological screening of urticaria.
方法采用酶免疫分析法对40例复发性口腔阿弗它溃疡的患者、21例健康人进行食物过敏原的检测。
Methods 40cases with RAU and 21 health people were detected by IVT enzyme immunoassay with food allergens.
本文采用酶免疫分析法测22例缺氧缺血性脑病（HIE患儿血清中神经元特异性烯醇化酶（NSE的变化。
Serum samples taken from 22 patients with hypoxia-ischemia encephalopathy (HIE were determined with enzyme immunoassay using a kit for the measurment of NSE.
采用电化学发光免疫分析法检测血清CA125、CA153、CA199，采用微粒子酶免疫分析法检测血清scc。
Electrochemical luminescence immunoassay method was used to detect CA153, CA199 and CA125, and particle enzyme immunoassay method was used to detect SCC.
方法：采用化学发光酶免疫分析法检测5 0例重症感染新生儿及4 0例正常健康儿童血清IL6、IL8水平。
Methods: Serum IL 6 and IL 8 were measured with chemiluminescent enzyme immunoassay in 50 newborns with severe infection and in 40 healthy infants.
采用光度酶联免疫分析法（ELISA）检测胰腺正常组、对照组、干预组中的LN的含量。
The Laminin(LN)level in normal group, control group and interference group was detected by using enzyme immunoassay(ELISA).
方法：采用放射免疫分析法和硝酸还原酶法测定20例HIE新生儿和20例正常新生儿血浆et与血清NO水平。
Methods: Plasma et levels were determined by using a radioimmunoassay kit, serum no levels were determined by using a nitrate reductase method in 20 newborn with HIE and 20 normal newborn.
方法采用固相酶联免疫分析法检测75例过敏性紫癜患者血清IL-18、VEGF水平。
Methods An enzyme-linked immunosorbent assay(ELISA)was utilized to detect the levels of IL-18, VEGF in serum of 75 patients with HSP.
目的：了解磁珠酶联免疫分析法检测人类白细胞抗原-B27的准确性、敏感性和特异性，探讨其在强直性脊柱炎辅助诊断中的价值。
OBJECTIVE: To explore the accuracy, sensitivity and specificity of IMS-ELISA for detecting HLA-B27 and its value in the auxiliary diagnosis of ankylosing spondylitis.
采用免疫组化方法、半定量逆转录聚合酶链反应（RT-PCR）和免疫印迹分析法检测COX-2、CDKN2A在组织中的表达。
The expression of COX-2 and CDKN2A was determined by immunohistochemistry, semi-quantitative reverse transcription polymerase chain reaction (RT-PCR) and Western blot.
采用免疫组化方法、半定量逆转录聚合酶链反应（RT-PCR）和免疫印迹分析法检测COX-2、CDKN2A在组织中的表达。
The expression of COX-2 and CDKN2A was determined by immunohistochemistry, semi-quantitative reverse transcription polymerase chain reaction (RT-PCR) and Western blot.
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