目的探讨急性一氧化碳中毒后迟发性脑病(DEACMP)患者脑神经元的损伤。
Objective To explore neuronal damage in patients with delayed encephalopathy after acute carbon monoxide poisoning (DEACMP).
目的：围产期缺氧缺血性脑损伤是导致儿童神经系统发育障碍及后遗症的严重疾病，已知脑损伤后迟发性神经元死亡以凋亡为主。
Objective: Perinatal hypoxic-ischemic brain injury is an important cause of neurodevelopmental impairment and disability. It's well-known that the main death 'form of neuron after HIBD is apoptosis.
目的：围产期缺氧缺血性脑损伤是导致儿童神经系统发育障碍及后遗症的严重疾病，已知脑损伤后迟发性神经元死亡以凋亡为主。
Objective: Perinatal hypoxic-ischemic brain injury is an important cause of neurodevelopmental impairment and disability. It's well-known that the main death 'form of neuron after HIBD is apoptosis.
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