结论：海马锥体细胞的迟发性坏死是缺血性脑血管病致痴呆的病理学基础。
CONCLUSION: Delayed necrosis of hippocampal pyramidal cells may be the pathological basis of ischemia cerebral vascular dementia.
缺血再灌注组脑皮层及海马CA1区苏木精-伊红染色示典型的迟发性神经元坏死。
The cells stained with haematoxylin and eosin displayed typical tardus neuron cellular necrosis in pallium and hippocampal CA1.
背景：脑缺血再灌注对中枢神经系统的影响，除了急性期的细胞坏死，还有迟发性的神经元凋亡。
BACKGROUND: Cerebral ischemia and reperfusion affects not only cellular necrosis at acute stage, but also delayed neuronal apoptosis in central neural system.
目的探讨低压电触电引起心肌坏死导致迟发性死亡的关系。
Objective to discuss the relation ship between low voltage electric shock and tardive death due to myocardial necrosis.
本实验结果揭示乳酸在短暂性脑缺血后海马迟发性神经元坏死（DND）发生中起着重要作用。
These results suggest that lactate plays an important role in the development of DND in hippocampus following transient forebrain ischemia in gerbils.
本实验结果揭示乳酸在短暂性脑缺血后海马迟发性神经元坏死（DND）发生中起着重要作用。
These results suggest that lactate plays an important role in the development of DND in hippocampus following transient forebrain ischemia in gerbils.
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