多囊卵巢综合征患者临床表现为：不孕、多毛，同时有高胰岛素血症，肥胖，高血压及血脂障碍。
Patients with PCOS have the clinical features of hirsutism and infertility; however, they also exhibit hyper insulinemia, obesity, hypertension, dyslipidemia.
目的研究血管性认知功能障碍(VCI)患者血脂代谢的变化。
Objective To investigate the change of metabolism of serum lipids in patients with vascular cognitive impairment (VCI).
结论缺血性中风尿微量蛋白增高与高血压分期、血脂水平及脂质代谢障碍的类型有关；
Conclusion Urine microproteins increasing in ischemic apoplexy is relative to stages of hypertension, serum lipid level and types of lipid dysbolism.
使这种情况复杂化的是，现实中还存在许多妨碍人们控制血压、血脂和血糖水平的障碍。
What makes this more complex is the reality that many other obstacles hold people from keeping their own blood pressure, lipids and blood glucose levels in check.
艾滋病毒是通过相关的免疫激活，慢性炎症，凝血功能障碍，和血脂紊乱机制，促进动脉粥样硬化形成。
HIV is known to promote atherosclerosis through mechanisms related to immune activation, chronic inflammation, coagulation disorders, and lipid disturbances.
目的：探讨青年抑郁障碍患者是否存在血脂代谢的异常。
Objective: To study if there exists abnormal metabolism of lipid in young depression patients.
结论空腹血糖水平与血脂异常程度相关，脂代谢障碍是2型糖尿病血管并发症的重要因素。
Conclusion FBS was correlated with blood lipid. Obstruction of blood lipid metabolism was an important faiot of type - 2 diabetes mellitus with angiopathy.
铜元素——可能导致溶血症，黄疸，血脂病变，加速氧化衰老，肾功能障碍甚至死亡。
Copper - Hemolysis, jaundice, changes in lipid profile, oxidative stress, renal dysfunction and even death.
目的了解脑卒中患者认知障碍的发生率并探讨高血压、高血脂、交流频率对其影响。
Objective to explore the incidence rate of cognitive deficits among stroke patients, and to explore the influence of hypertension, hyperlipemia and exchange-frequency on cognitive deficits.
目的研究轻度认知功能障碍(MCI)患者血脂代谢的变化。
Objective To study the change of metabolism of serum lipids in patients with mild cognitive impairment (MCI).
糖尿病的这些作用在血脂代谢障碍的小鼠的表现是不明显的，这表明血糖和血脂可能通过相似的机制而引起病变损害。
These effects of diabetes are masked in severely dyslipidemic mice, suggesting that the effects of glucose and lipids on lesion initiation might be mediated by similar mechanisms.
糖尿病的这些作用在血脂代谢障碍的小鼠的表现是不明显的，这表明血糖和血脂可能通过相似的机制而引起病变损害。
These effects of diabetes are masked in severely dyslipidemic mice, suggesting that the effects of glucose and lipids on lesion initiation might be mediated by similar mechanisms.
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