目的建立气管注入细菌致兔急性肺损伤(ALI)模型，为研究细菌致ali的致病机制及其治疗提供理论依据。
Objective: to establish the rabbit model of acute lung injury (ALI) by intratracheal instillation of Escherichia coli, and to study the pathogenesis of bacteria-induced ALI.
目的探讨胆红素对内毒素致急性肺损伤(ALI)的保护作用及其可能机制。
ObjectiveTo investigate the effects of of bilirubin on LPS-induced acute lung injury (ALI) in rats and its possible mechanisms.
目的探讨分泌型白细胞蛋白酶抑制剂（SLPI）对内毒素（LPS）致新生大鼠急性肺损伤（ALI）的抗感染作用。
Objective To investigate the anti-inflammatory effects of secretory leukocyte protease inhibitor(SLPI) on newborn rats with lipopolysaccharide(LPS)-induced acute lung injury(ALI).
方法：采用油酸合并脂多糖“两次打击”致大鼠急性肺损伤模型，观察鹅不食草挥发油对大鼠急性肺损伤的保护作用。
Method: the "Two-hit" animal model of acute lung injury in rats was induced by oleic acid and lipopolysaccharide, to observe the protective effect of VOCM on acute lung injury in rats.
目的探讨亚低温和高温对内毒素致急性肺损伤(ALI)大鼠肺微血管通透性的影响。
Objective to investigate the effects of mild hypothermia and hyperthermia on endotoxin-induced acute lung injury (ALI).
目的探讨LPS致新生大鼠肺损伤以至于肺出血的发病机制。
Objective To investigate the pathophysiology of acute lung injury in newborn rats induced by lipopolysaccharide.
酵母多糖活化的血浆可致山羊肺微血管壁受损，缺氧（模拟4000米高原）并未加重其损伤。
Thereafter hypoxic pulmonary pressor response was inhibited. ZAP could injure goat lung, hypoxia (4000m-simulated hig…
酵母多糖活化的血浆可致山羊肺微血管壁受损，缺氧（模拟4000米高原）并未加重其损伤。
Thereafter hypoxic pulmonary pressor response was inhibited. ZAP could injure goat lung, hypoxia (4000m-simulated hig…
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