但却指出大脑在这些条件下大体是抵抗自噬现象的。
But it was thought that the brain was largely resistant to autophagy under these conditions.
第7天后施万细胞内自噬泡数量开始减少。
After day 7, t he number of autophagic vacuoles began to diminish.
用高血糖素处理肝脏，会导制自噬作用增加。
第7天后许旺氏细胞内自噬泡数量开始减少。
From day 7on- wards the number of autophagic vacuoles began to diminish.
自噬与凋亡在细胞死亡过程中的关系十分密切。
There is a close relationship between autophagy and apoptosis during cell death.
在凋亡缺陷的肿瘤细胞中，自噬延长了生存期。
In tumour cells with defects in apoptosis, autophagy allows prolonged survival.
帕金森病相关神经变性中自噬溶酶体途径的作用。
The role of autophagy-lysosome pathway in neurodegeneration associated with Parkinson's disease.
当研究人员阻断自噬过程后，然而，那些饥饿信号也被阻断了。
When the autophagy was blocked by the researchers, though, those hunger signals were blocked.
结论受体介导内吞和自噬同凋亡之间存在一定的关系。
Conclusion Receptor-mediated endocytosis of ConA-HRP is associated with autophagy and apoptosis.
线粒体介导了死亡信号通路中自噬与凋亡的相互作用。
There is a mutual regulation between apoptosis and autophagy in death signaling process mediated by mitochondria.
目的：探讨大鼠坐骨神经变性轴突清除中的自噬作用。
Objective: to investigate the autophagic role of neuron axon during regeneration of rat sciatic nerves.
本文对自噬在肿瘤发生、转移与治疗中的作用进行综述。
This article summarizes the role of autophagy in Tumorigenesis, Metastasis and Therapy.
自噬可减轻炎症反应，同时也允许清除导致感染的微生物。
Autophagy reduces inflammation but at the same time permits the clearance of microbial infection.
本文对自噬的过程及其在细胞存活和死亡中的作用作一综述。
This review focuses on the processes of autophagy and its roles in cell survival and cell death.
自噬对于巨噬细胞清除尘粒和损伤的线粒体具有重要作用。
Autophagy plays an important role in cleaning dust particles and the injured mitochondria.
结论：尘粒沉积可引起巨噬细胞的自噬功能增强，并可出现凋亡。
CONCLUSION: Deposition of dust particles induces enhancement of autophagic activities and apoptosis of macrophages.
现在这个观念已被使用在细胞和生物机体的自噬基因缺陷之评估当中。
This concept has now been evaluated using cells and organisms deficient in autophagy genes.
今后的研究将决定自噬途径是否及如何操控外伤性脑损伤后的恢复改善。
Future studies will determine whether and how manipulation of the autophagy pathway improves post-TBI recovery.
目的了解肝纤维化和肝切除时同肝脏再生并行的自噬现象的表现和差别。
Objective To evaluate the characteristics of autophagy in fibrotic and postoperative remnant liver.
例如，自噬功能障碍与癌症，神经退行性病变，微生物感染和老化相关。
For example, autophagic dysfunction is associated with cancer, neurodegeneration, microbial infection and ageing.
真核细胞使用自噬和泛素蛋白酶体系统，作为它们主要的蛋白质降解通道。
Eukaryotic cells use autophagy and the ubiquitin-proteasome system as their major protein degradation pathways.
本研究显示，自噬途径在外伤性脑损伤的后的脑受伤区域内有明显的变化。
This study shows that the autophagy pathway is markedly changed in vulnerable brain regions after TBI.
然而，不可能确定自噬空泡的增加是否是一种适应性反应或细胞死亡的预兆。
However, it is not possible to determine whether the increase in autophagic vacuolization is an adaptive response or a harbinger of cell death.
结论：超微结构变化提示感光细胞能量代谢系统和(或)自噬系统功能障碍。
CONCLUSIONS: These changes suggested that self energizing system and self engulfing system of the photoreceptors were disfunctioned.
程序性细胞死亡有两种，包括凋亡和自噬性死亡，二者具有不同的形态学特征。
There are two morphologically distinctive forms of programmed cell death, apoptosis and autophagic cell death.
结论线粒体自噬可能为组织的损伤修复提供某种分子信号，促使新的线粒体再生。
Conclusion (Mitochondrial) autophagy may release some molecular signals for wound healing so as to accelerate sufficiently the regeneration of newly formed mitochondria.
超微结构与生化结果清楚的显示，TBI之后，自噬路径在神经元内被显著激活。
The ultrastructural and biochemical results clearly show that the autophagy pathway is significantly activated in neurons after TBI.
自相矛盾的是，自噬缺陷与肿瘤发生的呈正相关，但这背后的机制仍没有被明确。
Paradoxically autophagy defects are associated with increased tumorigenesis but the mechanism behind this has not been determined.
自相矛盾的是，自噬缺陷与肿瘤发生的呈正相关，但这背后的机制仍没有被明确。
Paradoxically autophagy defects are associated with increased tumorigenesis but the mechanism behind this has not been determined.
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