目的是观察大鼠局灶性脑缺血后小脑失联络现象及中药川芎嗪的干预作用。
The objective is to observe the crossed cerebellum diaschisis after focal cerebral ischemia and the interventional effects of ligustrazine.
目的介绍脑缺血治疗药物的研究进展。
OBJECTIVE To introduce progress in drugs for cerebral ischemia therapy.
目的探讨EEG对脑缺血的早期诊断价值。
Objective To explore the value of EEG in early diagnosis of cerebral ischemia.
证实了急性脑缺血对脑组织造成严重损害。
说明慢性脑缺血影响了大鼠的空间学习记忆。
It suggests that chronic brain ischemia affects rat spatial learning and memory.
脑缺血后的血管生成被一系列的血管因子调节。
Angiogenesis after cerebral ischemia was regulated by a series of vascular factors.
局灶性脑缺血后亦可增加海马齿状回内神经发生。
Focal ischemia also could raise the neurogenesis in hippocampus dentate gyrus.
这可能是缺血预处理诱导脑缺血耐受形成的机制之一。
This may be one of the possible mechanisms of cerebral ischemic tolerance by ischemic preconditioning .
方法阻断大鼠一侧大脑中动脉造成局灶性脑缺血模型。
Methods the model of cerebral ischemia was established by the unilateral middle cerebral artery occlusion in the rats.
而阿魏酸钠是否能增强脑缺血预处理的神经保护作用。
Whether does sodium ferulate enhance the nerve protection of IP brain to not.
目的探讨三七三醇皂苷对局灶性脑缺血损伤的保护作用。
Objective To study the protective effects of PTS on focal cerebral ischemia in rats.
结论采用上述方法可以成功建立血瘀性脑缺血大鼠模型。
Conclusion the methods can successfully establish the brain ischemia rat model combining blood stasis.
通过经颅阻断大鼠大脑中动脉制成局灶性脑缺血动物模型。
A model of cerebral infarction was established by occluding the left middle cerebral artery (MCA) transcranially.
目的观察人参果皂苷注射液对大鼠实验性脑缺血的保护作用。
Objective To observe the protective effect of ginseng fruit saponins injection on experimental cerebral ischemia in rats.
目的探讨短暂性脑缺血发作(TIA)与颅内外血管狭窄的关系。
Objective To investigate the relationship between transient ischemic attack (TIA) and intracranial or extracranial artery stenosis.
目的探讨CT脑灌注成像在短暂性脑缺血发作(TIA)的应用。
Objective To explore the application of ct perfusion imaging on transient ischemic attacks (TIA).
前言：目的：研究中风康对大鼠局灶性脑缺血后再灌注损伤的影响。
Objective: to study the pharmacological effects of Zhong Feng Kang on model mice with cerebral ischemia and after restoration of blood flow.
结论：EAAC 1反义寡核苷酸对急性脑缺血损伤有神经保护作用。
CONCLUSION: EAAC1 antisense oligonucleotide has the neuroprotective effect against acute cerebral ischemia injury.
采用大鼠反复性短暂脑缺血再灌注模型，观察丹皮酚的抗脑缺血作用。
Protcective effect of paeonol was observed in the repeated cerebral ischemia-reperfusion model in rats.
本实验提示巴曲酶对脑缺血再灌流损伤所引起的细胞凋亡有抑制作用。
The present study strongly indicate that batroxobin inhibits apoptosis induced by cerebral ischemia reperfusion injury.
大鼠脑缺血急性期脑甲状腺激素受体向上调节为机体的重要代偿反应。
The results indicated the upward regulation of TR in rat brain during acute cerebral ischemia is a compensatory response, which allows the maximum binding of thyroid hormones.
结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
另一种通俗的理论持相反观点，它认为是大脑缺血，或叫做脑部贫血，诱发睡眠。
Another popular theory took the opposite view, claiming that a lack of blood to the brain, or cerebral anemia, induced sleep.
目的研究短暂局灶性脑缺血预处理对再次脑缺血后微血管损伤及脑水肿的保护作用。
Objective To investigate the protection of brief focal cerebral ischemic preconditioning against microvascular injury and brain edema after subsequent severe focal ischemia.
该结果支持了S100 B可作为蛛网膜下动脉瘤破裂出血患者脑缺血的代用标记物。
This result supports the use of S100B as a surrogate marker for brain ischemia in patients with subarachnoid aneurysmal hemorrhage.
方法建立大鼠急性脑缺血再灌流损伤模型，用原子分光光度仪检测脑组织电解质含量。
Methods Cerebral ischemia reperfusion injury model was produced in rats. The electrolyte contents were measured with atomic absorption spectrophotometer(AAS).
方法建立大鼠急性脑缺血再灌流损伤模型，用原子分光光度仪检测脑组织电解质含量。
Methods Cerebral ischemia reperfusion injury model was produced in rats. The electrolyte contents were measured with atomic absorption spectrophotometer(AAS).
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