但是槟榔碱单体对INS - 1胰岛细胞的研究尚未见报道。
However, so far, there was little research of arecoline on INS-1 cells.
目的探索用简化的单一密度梯度法建立快速纯化大鼠胰岛细胞的方法。
Objective To establish an rapid and simple discontinuous density gradient method for purification of islet cells from rat pancreas.
文章就肝卵圆细胞及其定向诱导分化为胰岛细胞的相关研究作一综述。
This article reviews the research progress of hepatic oval cells and differentiation into pancreatic cells.
球形脂联素可拮抗胰岛细胞的脂性凋亡，从而发挥了其调节糖脂代谢的作用。
Adiponectin can counteract the lipoapoptosis in islet cells, and than plays a role in regulating lipid metabolism.
胰岛散布在胰腺内，无固定的形态，胰岛细胞的数量与类型因胰岛的大小而异。
The pancreas islet, without fixed form, scatter in the pancreas. The number and type of pancreatic endocrine cells change following the size of pancreatic islet.
即使不是这样，随葡萄糖而来的胰岛细胞的毁坏也许跟胰岛素抵抗的形成没有关系。
Even if it is not, the destruction of pancreatic cells that comes with diabetes may be unrelated to the development of insulin resistance.
结论为各种胰岛细胞的实验研究建立了快速分离纯化胰岛细胞的方法，纯化的胰岛细胞功能良好。
Conclusion We have established a simple and rapid rat islet purification method for islet study, and the purified islet cells remain full function.
然而，拥有又功能胰岛细胞的个体能够提高对糖尿病的控制能力，尽管他们仍然需要注射胰岛素。
However, individuals with functioning islets had improved control of their diabetes, even though they still needed to take insulin shots.
培养液中葡萄糖浓度的变化对胰腺细胞中的OX1R的表达没有显著影响，但会影响胰岛细胞的增殖功能。
The glucose fluctuation would affect the pancreas islet cell proliferation in vitro, but it does not induce the change of OX1R.
患病儿童医院和卡尔加里大学的研究人员假设，如果没有此项调控系统，胰岛细胞的正常功能变得不到发挥。
The researchers at the Hospital for Sick Children and the University of Calgary have postulated that without this control system normal function of islet cells cannot thrive.
高浓度的葡萄糖、糖皮质激素、脂肪酸可抑制PDX-1蛋白的表达，从而抑制胰岛细胞的胰岛素分泌功能。
Exposure of islet cells to high concentration of glucose, dexamethosone and palmitate can reduce the expression of PDX-1, therefore affects the insulin synthesis of islet cells.
患有更严重类型的糖尿病患者其胰岛素分泌细胞存在问题。
Sufferers from the more severe form of diabetes have faulty insulin-producing cells.
例如，某些蛋白质的过量会导致细胞异常增殖并癌变；胰岛素蛋白质的缺乏会导致糖尿病。
For example, an excess of certain proteins can cause cells to proliferate abnormally and become cancerous; a lack of the protein insulin results in diabetes.
目的是介绍小儿胰岛细胞增生症的诊断和治疗经验。
The objective is to present the experience in the diagnosis and treatment of children's nesidioblastosis.
然而，有糖尿病的人，胰腺要么生成一点或根本没有生成胰岛素，或者是细胞没有正确的反应胰腺分泌的胰岛素。
In people with diabetes, however, the pancreas either produces little or no insulin, or the cells do not respond appropriately to the insulin that is produced.
尽管胰岛素对于这些细胞的影响还没有被很清楚的了解，但科学家知道成骨细胞中存在胰岛素受体却已经是事实了。
Scientists have known for some time that the bone-building osteoblasts have insulin receptors, although the effect of insulin on these cells was not well understood.
但与正常小鼠不同，那些使用改变巨噬细胞的表明免疫炎症症状，像在胰岛素产生的变化那样，高水平免疫化学物，和在它们腹部脂肪中的巨噬细胞。
But unlike the normal mice, those with altered macrophages showed no signs of inflammation, such as changes in insulin production, high levels of immune chemicals, and macrophages in their belly fat.
胰岛素是绑在细胞上的胰岛素受体上的。
Insulin binds to what are called insulin receptors on your cells.
为了使这些细胞变成胰岛分泌细胞，他们将这些细胞暴露与化学试剂通过三周的特定过程诱导分化。
To convince the cells to become insulin producers, they exposed them to chemicals that stimulate differentiation at specific times over the course of about three weeks.
当我们吃的时候，胰岛将自动的分泌足够的胰岛素，这些胰岛素将葡萄糖从血液中移进我们的细胞。
When we eat, the pancreas automatically produces the right amount of insulin to move glucose from blood into our cells.
1型糖尿病，胰腺中的某些细胞在生产胰岛素时逐渐恶化，导致胰岛素缺乏。
In type 1 diabetes, certain cells in the pancreas gradually get worse at producing insulin, leading to insulin deficiency.
在将老鼠的肝细胞转变为制造胰岛素的细胞后，科学家们现在断言，糖尿病的治疗将会取得进展。
But now scientists claim a cure could be developed after cells in the liver were converted to insulin producers in research on mice.
以前曾已将肝细胞转变为制造胰岛素的细胞，但科学家明白该技术还是首次，他们也相信最终该技术将应用于人类的糖尿病治疗。
Liver cells have been coverted to insulin producers before but this is the first time scientists have understood the technique and believe eventually could lead to treatment for humans.
它们可不附加任何基因而被诱导为胚胎样细胞，然后变为胰岛素分泌细胞，加里卡诺说此方法比其它方法更少的诱发恶性肿瘤。
They can be coaxed into an embryonic-like state without any additional genes, and after becoming insulin-producing cells, they produce less cancer than other methods, according to Gallicano.
问题关键在于找到正确的诱导干细胞分化成心脏细胞，神经细胞，制造胰岛素的细胞或者其他细胞的生长因子的正确方法。
The key lay in finding just the right recipe of growth factors and nutrients to induce a stem cell to become a heart cell, a neuron, an insulin-making cell or something else.
当身体对胰岛素产生抵抗力时，细胞便不能得到所需的能量。
When your body is resistant to insulin, your cells can't get the energy they need.
有2型糖尿病时，细胞抵抗胰岛素的作用，使葡萄糖难以进入细胞。
In type 2 diabetes, the cells resist insulin's action, so glucose has trouble getting into the cells.
有2型糖尿病时，细胞抵抗胰岛素的作用，使葡萄糖难以进入细胞。
In type 2 diabetes, the cells resist insulin's action, so glucose has trouble getting into the cells.
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