• 目的观察发育过程中脑神经干细胞增殖迁移分化特点

    Objective To evaluate the characteristics of proliferation, migration and differentiation of neural stem cells in the brain of rat embryo during development.

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  • 结论血清igfbp - 1作为生长抑制因子参与正常胎鼠发育IUGR发生。

    Conclusions Serum IGFBP-1 participates in the growth of normal and IUGR rat fetuses as an inhibiting factor.

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  • 结果:康乃精颗粒一般状况体重变化发育分娩新生以及子代大发育状况均无任何毒性作用。

    Results: KNJ had no toxicologic effect in the general status, body weight change, and embryonic rat development, maternal rat delivery, newborn rat as well as the development status of rat.

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  • 目的探讨胰岛素样生长因子结合蛋白- 1 (IGFBP - 1)正常发育胎鼠宫内发育迟滞(IUGR)发生中的作用

    Objective To explore the role of IGFBP-1 in the normal growth and intrauterine growth retardation (IUGR) in rat fetuses.

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  • 然后研究者发育中的胎鼠暴露损伤

    Then the researchers exposed the brains of the developing fetuses to injury.

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  • 目的了解蚂蚁宫内发育迟缓(IU GR)大脑肝脏作用

    Objective To investigate the effect of ant powder on the brain, liver and placenta of fetal rat with IUGR.

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  • 观察母生长发育情况。

    The development of maternal rats and embryos were observed.

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  • 目的了解左旋精氨酸被动吸烟所致大宫内发育迟缓发育影响通过NOMDASOD关系探讨其作用机制。

    To explore the effects of L-arginine on the brains of fetal rats with IUGR and the relationship with the levels of NO, MDA and SOD.

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  • 目的研究宫内生长受限(FGR)发育远期学习记忆能力变化

    Objective: To study the changes of cerebral development and learning and memory ability of fetal rats with intrauterine fetal growth restriction (FGR) after birth.

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  • 目的从分子水平探讨叶酸缺乏FD发育影响。

    Objective: To verify the effect of maternal folate deficiency (FD)on fetal rat brain development, and the change of DNA methylation.

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  • 目的:探讨叶酸缺乏宫内发育影响研究叶酸缺乏孕子代脑组织基因组dna甲基化水平的改变叶酸缺乏造成脑发育障碍提供分子水平依据。

    Purpose: To verify the effect of folate deficiency on fetal rats intrauterine brain development, the change of DNA methylation in fetal rats brain with maternal folate deficiency was studied.

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  • 电厂下风侧居民区内收集大气悬浮微粒,二氯甲烷提取浓缩制成受试物,给孕腹腔注射不同剂量,观察对小发育的影响。

    Fallout particulates from ambient air were collected in an leeward residential area of a power plant. Then, were extracted with dichloromethane, concentrated and redisolved in olive oil.

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  • 率均有所下降胎鼠生长发育受到干扰。

    Both the rate of dead fetuses and of the monstra were decreased but the development of the fetuses was disturbed.

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  • 实验组胎鼠的身长、体重发育指标均有显著降低

    The mean body weight, average body length, tail length significantly decreased.

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  • 我们研究了维太明、EDTA孕期和哺乳期暴露和幼生长发育原卟啉和血压影响

    We studied the effects of VITALCARE, EDTA and lead exposure on lead toxicity, fetal and neonatal growth, erythropoiesis and blood pressure during pregnancy and lactation in rats.

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  • 试验证明,孕期哺乳期,维太明、EDTA暴露的原啉和血压以及和幼生长发育均可产生影响

    The results demonstrate that VITALCARE EDTA and lead exposure interact in rats to influence maternal blood pressure, erythropoiesis, and fetal and neonatal growth during pregnancy and lactation.

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  • 试验证明,孕期哺乳期,维太明、EDTA暴露的原啉和血压以及和幼生长发育均可产生影响

    The results demonstrate that VITALCARE EDTA and lead exposure interact in rats to influence maternal blood pressure, erythropoiesis, and fetal and neonatal growth during pregnancy and lactation.

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