采用免疫组化的方法检测52例肺癌组织MRP的表达。
Methods Immunohistochemistry was used to detect the expression of MRP in 52 patients.
结论CYP2A6突变基因在肺癌组及对照组中的频率分布没有差异。
Conclusion There is no difference in frequency of CYP2A6 gene deletion between lung cancer cases and controls.
在对照肽(左)或抗原特异性多肽(右)存在的条件下使用FAk抗体对石蜡包埋的人肺癌组织进行免疫组化分析。
Immunohistochemical analysis of paraffin-embedded human lung carcinoma, using FAK Antibody in thre presence of control peptide (left) or antigen-specific peptide (right).
方法采用电化学发光法(ECLIA)分别对肺癌组、肺部良性疾病组患者及健康人的血清肿瘤标记物浓度进行检测分析。
Methods Serum tumor markers of lung cancer group, benign disease group and health control group were detected by chemiluminescence immunoassay (ECLIA).
方法应用免疫组化的方法检测新辅助化疗组和对照组肺癌组织中MMP—2和TIMP—2蛋白的表达，并分别计算其表达比率。
Methods The test group and control groups' expression of MMP-2, TIMP-2 were detected by immunohistochemical method, then the ratios of MMP-2/TIMP-2 were calculated.
方法用PCR-RFLP技术分析了原发性肺癌组和住院对照组（各163例）的CYP1A1、GSTM1基因的多态性、基因型分布频率和交互作用。
Methods CYP1A1 and GSTM1 genetic polymorphisms were determined by PCR-RFLP in 163 lung cancer cases and healthy controls respectively.
结果显示肺癌组病变处支气管呈不规形扁平，刀鞘与鼠尾状狭窄，造影缺损、阻塞、管壁僵硬如枯技状；肺泡癌有肺泡充盈不良、缺损和造影剂不能进入肺泡。肺炎组病变支气管均充盈显影。
The results revealed that in lung cancer the lesions showed irregular flattening, sword—sheath shaped or rat—tail shaped stricture, filling defects, obstruction and rigidity of the bronchial wall;
研究员发现顺铂可以有效对抗这两种小鼠(Kras和p53基因突变组)的肺癌，即使它在那些仍旧有有功能的p 53的小鼠更有效。
The researchers found that cisplatin was effective against lung tumors in both sets of mice, though it was more potent in mice that still had functional p53.
实际上，这并不意味着此处所讨论的基因或基因组会造成肺癌，而是说它们放大了吸烟的影响而非吸烟的强度。
That does not mean the gene or genes in question actually cause lung cancer. Rather, it means that they amplify the effects of smoking instead of the amount of smoking.
对肺癌基因组的研究已获得了某种有用的信息。
Already the lung cancer genome is yielding useful information.
桑格研究所开展的这项研究从一名肺癌患者和一名皮肤癌患者身上分别提取肿瘤细胞组织和正常细胞组织，运用新型高效的DNA测序技术对其基因组进行了全面破解。
The Sanger Institute studies used powerful new DNA sequencing technologies to decode completely the genome of both tumour tissue and normal tissue from a lung cancer and a malignant melanoma patient.
每年致3.45万人死亡的肺癌，其基因组包含2.3万个突变；而每年致2000人死亡的皮肤癌，其基因组包含3.3万个突变。
The lung cancer genome, which kills 34,500 people a year, contained more than 23,000 mutations, the melanoma, which kills 2,000 people a year, more than 33,000.
尽管导致患者死亡的原因主要是肺癌，但是贝伐单抗组的14名患者以及对照组的2名患者却死于药物毒性。
Causes of death were predominantly due to lung cancer, but 14 patients in the bevacizumab group and 2 in the control group died as a result of drug toxicity.
方法93例肺癌患者，根据不同治疗方案随机分为第一、第二和第三组。
Methods 93 patients with lung cancer were randomly divided into three groups based on different regimens.
方法采用端粒重复序列扩增法一酶联免疫（PCR-TRAP）检测70例肺癌手术患者癌组织中的端粒酶活性，并与70例肺部良性疾病患者对照组的检测结果进行比较。
Methods Use PCR-TRAP detection of 70 cases of lung cancer patients' telomerase activity in cancer tissue. and 70 patients with benign lung disease and the results were compared.
对2545名铬酸盐生产工人和5197名对照组工人进行了吸烟与肺癌发病关系的回顾-前瞻性流行病学调查研究。
Retrospective and prospective epidemiological investigations on relationship between smoking and lung cancer in 2545 workers of chromate production and 5197 control workers were carried out.
本研究共纳入170名肺癌病患和340名正常对照组。
This study included 170 lung cancer patients and 340 normal controls.
患者中的三组进行了测试：110名健康人，60例肺癌和慢性阻塞性肺疾病50，肺部呼吸道变窄。
Three groups of patients were tested: 110 healthy people, 60 with lung cancer and 50 with chronic obstructive pulmonary disease, a narrowing of the airways of the lungs.
结论：晚期肺癌和肺部良性疾病患者血浆纤维蛋白原水平与功能均高于正常对照，两组间差异有显著性。
Conclusion: Plasma fibrinogen concentration increases and its molecule function was abnormal in developing lung cancer and pulmonary benign disease. It was significantly difference in the two groups.
结果肺癌患者在焦虑、抑郁、愤怒、理智、情绪控制、乐观及社会支持方面均与对照组有显著性差异。
Results There were notable differences between the lung cancer group and the control group in the following aspects:anxiety, depression, anger, reason, mood control, optimism and social support.
在这项研究中，他们检测了76位人类基因组，发现了令人惊讶的转座因子出现频率，并发现它们在肺癌基因组中非常活跃。
For this study they examined the genomes of 76 people and found transposons were surprisingly prevalent; they also found they were very active in lung cancer genomes.
一组研究人员提出了一个诊断肺癌的好主意。
A team of researchers may have come up with a golden idea for diagnosing lung cancer.
肺癌晚期组丘脑及豆状核区域代谢增加。
The group of advanced lung cancer also showed the increased metabolism in thalamus and basal ganglion.
结论：肺癌纵隔淋巴结的转移形式与预后相关，跳跃式转移及转移组数多的病例预后更差。
Conclusions: Forms of mediastinal lymph node metastasis relates with prognosis of lung cancer, prognosis of cases with skip metastasis or more regional lymph node metastasis is worse.
目的：探讨非小细胞肺癌神经内分泌分化的免疫组化诊断。
Objective: to study the immunohistochemical diagnosis of neuroendocrine differentiation in surgically resected NSCLC.
肺癌的分析实验包括4，775个肺癌患者和2，835人组成的对照控制组，所有人都是经常吸烟者。
The lung cancer analysis included 4,775 lung cancer cases and 2,835 controls, all of whom were regular cigarette smokers.
方法：用免疫组化技术检测了52例非小细胞肺癌及转移淋巴结中ICAM-1的表达。
Methods:ICAM 1 Expression in 52 cases of primary and metastatic NSCLC were studies on cytostat sections by means of immunohistochemistry.
方法：运用端粒酶pcr -TRAP酶联免疫吸附试验(ELISA)测定法对30例肺癌患者及25例肺部良性疾病患者(对照组)的痰液进行端粒酶活性检测。
Methods: The sputa of 30 lung cancer patients and 25 benign lung disease patients (control group) were checked for telomerase activity with PCR-TRAP ELISA.
方法将56例小细胞肺癌患者以随机方式分为治疗组ce(卡铂、VP - 16)方案化疗并用力尔凡和对照组单用CE方案化疗。
Methods 56 patients with SCLC were randomly divided into treatment group and control group receiving ce (Carboplatin, Etoposide VP-16) regimen plus Lifein or ce regimen alone respectively.
方法将56例小细胞肺癌患者以随机方式分为治疗组ce(卡铂、VP - 16)方案化疗并用力尔凡和对照组单用CE方案化疗。
Methods 56 patients with SCLC were randomly divided into treatment group and control group receiving ce (Carboplatin, Etoposide VP-16) regimen plus Lifein or ce regimen alone respectively.
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