结论 肢体缺血再灌注可引起急性肺损伤。
Conclusion Reperfusion of acute ischemic extremites may produce structural change in lung.
结论ET - 1可能参与了肢体缺血再灌注损伤。
Conclusions ET-1 is perhaps participated in limb ischemia-reperfusion Injury.
肢体缺血再灌注后关节软骨及滑膜损伤的组织学观察。
Histological Observation of Articular Cartilage and Synovial Injury after Ischemia in the Animal Hind Limbs.
结果：肢体缺血再灌注后，肠道屏障功能明显受到损伤和破坏。
Result: After Lower limb ischemic reperfusion, the intestinal barrier function was severely damaged.
肢体缺血再灌注损伤是临床上研究热点，特别是病理改变的研究。
The limb ischemia reperfusion injury is the hot point in clinical study especially in pathological change.
目的通过动物实验观察肢体缺血再灌注损伤对肺组织形态学结构的影响。
Objective To observe pathology change of the lung after lower limb ischemic reperfusion injury.
大鼠肢体缺血再灌注后发生了肺损伤，静脉输注高渗盐水有一定的肺保护作用。
Lung injury occurred in rats after LIR and intravenous infusion of FIS has some lung protective effects.
方法：制作兔肢体缺血再灌注损伤动物模型，并分为对照组、再灌注组和治疗组。
Methods: the experimental models of ischemia and reperfusion injury of limb was produced in the rabbits which were divided into control group, reperfusion group and treatment group.
方法：制作兔肢体缺血再灌注损伤动物模型，实验分对照组、再灌注组和治疗组。
Methods: The experimental model of ischemia reperfusion injury of limb was produced in the rabbits which were divided into control group, reperfusion group and treatment group.
结论非创伤性肢体缺血预适应对心脏缺血再灌注损伤具有早期保护作用，能明显缩小心肌梗死面积。
Conclusion: the invasive limb IPC has an early protective effect on the myocardial ischemia-reperfusion injury and could decrease the myocardial infarct size.
结论非创伤性肢体缺血预适应对心脏缺血再灌注损伤具有早期保护作用，能明显缩小心肌梗死面积。
Conclusion: the invasive limb IPC has an early protective effect on the myocardial ischemia-reperfusion injury and could decrease the myocardial infarct size.
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