在显微镜下，肝脏是由肝小叶为单位的网状结构。
When viewed under a microscope, the liver is seen as large network of units called hepatic lobules.
肝动脉血栓形成是最可怕的一种肝移植术后并发症，会导致肝小叶广泛坏死、肝梗死和胆管狭窄。
Hepatic artery thrombosis is one of the most dreaded complications of liver transplant and can cause massive hepatic necrosis, hepatic infarcts and biliary strictures.
第18天以后，正常的肝小叶结构开始恢复。
After day 18, the normal liver lobule structures began to recover.
模型组大鼠肝小叶结构紊乱，有大量纤维组织沉积，炎症细胞浸润，肝细胞脂肪变性。
Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue inflammatory cell infiltration and fatty degeneration of hepatocytes.
病理基础是预处理毒性引起肝脏内肝小叶3区内的血窦内皮细胞以及肝细胞损害所致。
The basis of pathology of vod is thought resulting from injury of hepatocytes and endodermis cells surrounding the central veins in zone3of the liver acinus by regimen-related toxicity.
暗红色淤血区表示肝小叶中心区的红细胞积聚。
Note the dark red congested regions that represent accumulation of RBC's in centrilobular regions.
建模第10 ~12周，肝小叶结构更加紊乱，出现早期肝硬化迹象。
In the 10 ~ 12 weeks, a more disordered structure of hepatic lobule, early cirrhosis.
肝板多由双层细胞构成，肝小叶不明显。
虽然血管结构存在，但无可辨认的正常肝小叶结构。
There is no discernable normal lobular architecture, though vascular structures are present.
肝脏组织学特点为汇管区纤维增生明显，肝小叶结构正常。
The histologic characteristics include fibrous proliferation of interlobular portal areas , and normal structure of hepatic lobules.
对照组肝小叶结构和肝细胞状态正常，仅在中央静脉及汇管区可见少量胶原纤维。
In the control group, the structure of liver lobule was normal and a few of collagen fibers located in portal area and central vein.
脂肪性肝病是多种原因引起的病变主体在肝小叶、以肝细胞脂肪变性和脂肪蓄积过多为主的临床病理综合征。
Fatty liver disease (FLD) is a clinicopathological syndrome caused by a variety of reasons. The main lesions are in hepatic lobule with steatohepatitis and accumulation of triglycerides in the liver.
结果四氯化碳注射12周后，兔肝组织出现典型的假小叶，肝组织羟脯氨酸含量较正常组升高3.5倍。
Results 12 weeks after carbon tetrachloride administration, the rabbit liver developed typical pseudo-lobule, and the liver hydroxyproline content increased 3.5 times compared to normal control.
随着肝脏增大，肝小叶的形成，排列成由1 ~2层甚至单层的肝细胞索。
As the liver increasing and the liver lobule forming, 1-2 layers of cells arrange in the liver cell cords.
可见肝中央静脉的肝小叶中央区。
Here is the centrilobular portion of liver next to a central vein.
病理切片检查显示正常组的大鼠肝组织形态结构正常，肝小叶规则，无脂滴发现；
The pathology slice examination shows: the liver tissue organization of the rats in normal comparison group is normal and the hepatic lobules of them are well-arranged without liquid droplets;
ILD组肝内胶原纤维明显增生，假小叶形成。
The collagen fibrils had significant hyperplasia, and false liver acinus formed in liver of ILD.
肝在组织学上分为无数的肝小叶。
恢复期结束肝结构均正常。中剂量组：同对照组，可见肝小叶、肝索及门管区。
The recovery period conclusion liver structure was normal. in monitoring team: With control group, obviously hepatic lobe, liver rope disciple district.
恢复期结束肝结构正常。小剂量组：同对照组，可见肝小叶、肝索及门管区。
The recovery period conclusion liver structure was normal. small monitoring team: With control group, obviously hepatic lobe, liver rope disciple district.
若进一步引起肝小叶改建，假小叶和结节形成，则致肝硬化。
If it further causes the hepatic lobe reconstruction, pseudolobuli and tubercle take shape, then comes the liver cirrhosis.
肝静脉栓塞导致肝小叶中心性坏死、硬化与肝静脉-门静脉瘘。
However, embolization of hepatic vein resulted in central necrosis of the lobules, liver cirrhosis and formation of fistulas between the hepatic vein and the portal vein.
中央静脉位于小叶中央，肝细胞排列成肝细胞索肝板，以中央静脉为中心向周围呈放射状排列。
Passing through the center of lobules, a central vein is radiating and arranging by hepatic cord or plate. Interval the hepatic cords or masses are irregular and variable sinusoid.
实验组与对照组相比，肝小叶损伤区域缩小，肝细胞中脂滴减少，琥珀酸脱氢酶活性增强。
Compared to the control group, the necrotic area of hepatic lobule and fat droplets of hepatic cells were decreased and SDH activity was increased in the experimental group...
肝静脉栓塞导致肝小叶中心性坏死、硬化与肝静脉-门静脉瘘。
However, embolization of hepatic vein resulted in central necrosis of the lobules, liver cirrhosis and formation of fistulas between the hepatic vein and the portal...
图示中央静脉旁的肝小叶中央部，肝细胞体积缩小或缺氧坏死。
Here is the centrilobular portion of liver next to a central vein. The cells have reduced in size or been lost from hypoxia.
肝小叶小心性坏死和出血（1周内）；
Centrilobular necrosis and haemorrhage in the liver (in one week);
肝小叶小心性坏死和出血（1周内）；
Centrilobular necrosis and haemorrhage in the liver (in one week);
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