心肌细胞核染色质结构接近正常；
Nucleolus chromatin structure of cardiomyocytes was close to normal;
大鼠心肌细胞核采用差速离心和密度梯度离心分离提纯。
Velocity and isopyknic gradient centrifugation was employed to fractionate rat myocardial nuclei.
目的研究小鼠死后骨骼肌细胞核DNA降解与死亡时间的关系。
Objective To study the relationship between changes of DNA degradation and the postmortem interval.
结果：正常骨骼肌纤维横截面积较大，形态规则，肌细胞核紧贴于肌纤维的边缘；
Results:Cross section area of normal skeletal muscle fiber was large, morphous was regular, myocyte nucleus located in the edge of muscle fiber;
目的研究兔颈动脉平滑肌细胞核d NA含量随缺血时间而变化的关系，判断断指再植时限。
Objective To evaluate the DNA degradation in smooth muscle cell nuclei of carotid in rabbits during ischemia, and study the relationship between the degradation and ischemic time.
病理染色图象分析测定左室腔周长和面积、单位面积内心肌细胞核数、单位面积内胶原的含量；
Pathological staining and image analysis were used to determine the perimeter and the area of left ventricular cavity, and myocardial nuclei number and collagen content per unit area;
目的：探讨心肌缺血再灌注损伤时心肌细胞核钙含量与核孔复合体通透性的变化及其相互关系。
AIM: To investigate the changes in nuclear calcium content and permeability of nuclear pore complex in rat myocardium during ischemia reperfusion injury.
在分离的LV上以相同方向、均匀的切片，使用无偏倚的立体学方法，评估心肌细胞核总数、每个心肌细胞的细胞核数。
Isotropic, uniformly random sections were cut from theisolated LV, and the total number of myocyte nuclei and the average number of nuclei per myocyte were estimated using unbiased stereology.
结果心内膜下缺血心肌细胞核异常肥大、畸形，肥大畸形细胞核数目明显多于正常对照组，两者有明显差异（P <0 .0 1）。
Results The nuclear of ischemic myocardium was enlargement and abnormity under endocardium. The number of abnormal nucleus in ischemic myocardium was more than that in the normal controls(P<0.01).
结果发现，主动脉平滑肌细胞可表达细胞周期素d，其免疫反应产物主要位于细胞核内。
Results Rabbit smooth muscle cells could express cyclin d, the reactant of immune reaction was mainly located in nuclei.
在心肌细胞中，T3结合到细胞核中的甲状腺激素受体，其依次结合靶基因调节区中的甲状腺激素反应元件以调节转录。
In the cardiomyocyte, T3 binds to thyroid hormone receptors in the nucleus, which in turn bind to thyroid hormone response elements in the regulatory regions of target genes to regulate transcription.
在心肌细胞中，T3结合到细胞核中的甲状腺激素受体，其依次结合靶基因调节区中的甲状腺激素反应元件以调节转录。
In the cardiomyocyte, T3 binds to thyroid hormone receptors in the nucleus, which in turn bind to thyroid hormone response elements in the regulatory regions of target genes to regulate transcription.
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