缺血预适应表现为早期和延迟心肌保护。
Transient ischemia (preconditioning stimulus) not only triggers early preconditioning, but also induces delayed protection.
缺血预适应可明显改善亚细胞结构损害。
Ischemia preadaptation can evidently reduce subcellular structure injury.
心肌缺血预适应与细胞信号转导密切相关。
Myocardial ischemic preconditioning is related to cellular signal transduction.
结论缺血预适应的保护作用在老年人减弱。
Conclusion Protective effect of ischemic preconditioning decline in the elderly.
并且近、远期缺血预适应存在不同机制并有相加作用。
New and old preconditioning had different mechanisms with adding effect each other.
目的研究缺血预适应对老年心肌梗死范围和预后的影响。
To investigate the effects of myocardial ischemic preconditioning on size and prognosis of myocardial infarction in the aged.
目的探讨急性心肌梗死合并泵衰竭与心肌缺血预适应的关系。
Objective To ascertain the relationship between acute myocardial infarction combined pump exhaustion and myocardial ischemic preconditioning(MIP).
这种缺血预适应现象可以分为“早期保护”和“延迟保护”。
This ischemic preconditioning(IP) phenomenon can be divided into"early protect" and "delayed protect".
结论：心肌缺血预适应可以限制梗死面积扩大，降低死亡率。
Conclusions Ischimia precondition can not only constrict the area of AMI, but also decrease its mortality.
方法和结果——在体的缺血预适应能增加细胞质膜微囊的形成。
Methods and Results - Ischemic preconditioning in vivo increased the formation of caveolae.
方法采用高效液相色谱法测定缺血预适应过程中腺苷浓度变化；
Methods The changes of adenosine concentration after ischemia preconditioning were measured using high performance liquid chromatogram(HPLC) system.
【摘要】目的研究缺血预适应对心肌梗死范围及近期预后的影响。
Abstract Objective To study effects of preconditioning on size of myocardial infarct and short-term prognosis.
目的：探讨无创性肢体缺血预适应在提高小鼠抗应激能力方面的作用。
Aim: To explore the effects of noninvasive limb ischemic preconditioning on the anti-stress ability in mice.
缺血预适应的心肌保护作用是心脏释放内源性心血管活性物质所介导。
The cardioprotective effect of ischemic preconditioning may be mediated by endogenous cardiovascular active substances.
结论：梗塞前心绞痛发作对心肌产生的缺血预适应有明确的心肌保护作用。
Conclusion: the angina attack before infarction can produce the ischemic preconditioning on myocardium that provides myocardium with definite protection.
目的探讨缺血预适应(PC)心电高频能量的变化规律和缺血预适应的机制。
Objective To study variety of the high frequency energy and preconditioning (PC) mechanism in preconditioning.
结论缺血预适应可升高缺血再灌注损伤后肾脏HSP70的表达，改善肾功能。
Conclusion Kidney ischemic preconditioning could increase the expression of HSP70 and decrease the injury of ischemia reprefusion.
目的探讨缺血预适应对急性心肌梗死Q_T间期离散度及室性心律失常的影响。
Objective To investigate effect of ischemic preconditioning on Q _ T dispersion(Q _ Td)and ventricular arrhythmia in acute myocardial infarction.
因此，笔者认为有必要借鉴缺血预适应现象，阐述其作用机制，弘扬针灸优势。
Therefore, it's very necessary to explain the mechanism of the effect and to develop advantages of acumoxi by using for reference of the phenomenon of ischemic precondition.
本实验室以前的研究发现脑缺血预适应过程中BER系统具有可逆性调控机制。
Our lab's studies have found that BER in the brain is a highly regulated process after ischemia-precondition.
方法采用实验性家兔缺血再灌注及缺血预适应模型，随机分成7组，每组8只。
Method By using experimental rabbits IR (Ischemia Referpusion) and IP (Ischemia Preadaptation) model, we freely divide rabbits into 7 groups (8 rabbits every group).
缺血预适应现象与再灌注后氧自由基生成增多、细胞内钙超载、细胞凋亡等有关。
It is often related with increasing oxygen free radical, calcium overload and cell apoptosis.
结论心肌梗死前的心肌缺血预适应对老年心肌梗死患者的心肌有一定的保护作用。
ConclusionIschemic preconditioning before mi has protective effect on myocardium of the aged against myocardial infarction.
目的探讨心肌缺血预适应（IP）对急性心肌梗死（AMI）临床及预后的近期影响。
Objective To investigate the short-term effects of ischemic preconditioning(IP) on clinical and prognosis in patients with acute myocardial infarction(AMI).
目的评价缺血预适应(IPC)防治猪急性心肌梗死(ami)再灌注后无再流的作用。
Objective To evaluate the effects of ischemic preconditioning (IPC) on myocardial no - reflow in a mini-swine model of acute myocardial infarction (AMI) and reperfusion.
越来越多的基础研究证明吸入麻醉剂在缺血预适应和缺血后适应中都具有心肌保护作用；
Various laboratory studies have demonstrated that inhalation anesthetics play a protective role in the myocardial ischemic preconditioning and postconditioning.
目的：观察缺血预适应(IPC)对猫体外循环(CPB)前后心肌细胞膜脂流动性的影响。
Objective: To evaluate the effect of ischemic preconditioning (IPC) on myocardial cell membrane fluidity in feline heart with cardiopulmonary bypass (CPB).
结果（1）缺血预适应组心肌酶水平与缺血再灌注组相比差异均极为显著（P<0.01）；
Results (1)Level of myocardium enzyme with ischemia reperfusion group is significantly different compared to ischemia preadaptation(P<0.01).
目的：探讨解偶联蛋白- 2 (UCP2)在心肌缺血预适应(IPC)心肌保护中的作用。
Objective: To discuss the role of uncoupling protein 2 (UCP2) in myocardium with ischemic preconditioning (IPC).
目的：探讨解偶联蛋白- 2 (UCP2)在心肌缺血预适应(IPC)心肌保护中的作用。
Objective: To discuss the role of uncoupling protein 2 (UCP2) in myocardium with ischemic preconditioning (IPC).
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