新生儿期发现缺血缺氧性脑病1例，颅内出血4例。
In neonatal stadium, 1 patient was found with hypoxia-ischemic encephalopathy, and 4 with cerebral hemorrhage.
目的：探讨纳洛酮对新生儿缺血缺氧性脑病的治疗作用。
Objective:To investigate the therapeutic effect of naloxone on ischemic hypoxic cerebropathy in neonates.
目的探讨高压氧疗法对缺血缺氧性脑病(HIE)的疗效。
Objective To discuss the curative effect of HIE treated with HBO.
目的探讨超声在新生儿缺血缺氧性脑病(HIE)的诊断价值。
Objective Explore the ultrasound in the neonatal hypoxic ischemic encephalopathy (HIE) the diagnostic value.
目的观察新生鼠缺血缺氧性损伤后皮层及海马神经干细胞的变化。
Objective To investigate the influence of hypoxic- ischemic(H-I) injury on the endogenous Neural Stem Cells (NSCs) of the cerebral cortex and hippocampus in the neonatal rats.
目标：确定窒息新生儿罹患新生儿缺血缺氧性脑病(HIE)的危险因素。
OBJECTIVE: to determine the risk factors for hypoxic-ischemic encephalopathy (HIE) in asphyxiated newborn infants.
结论：螺旋CT头颅检查对新生儿脑缺血缺氧性脑病的临床分度有重要意义。
Conclusion:Sprial CT is important in the detection of neonatal hypoxic_ischemic encephalopathy.
结论：纳洛酮在一定程度上能改善新生儿缺血缺氧性脑病的病情，促进其恢复。
Conclusion: Naloxone can improve the conditions of ischemic hypoxic cerebropathy in neonates to a definite degree and facilitates recovery.
方法回顾性分析3 2例缺血缺氧性脑病患者恢复期的临床特征及ADL康复疗效。
MethodsThe clinical features and ADL rehabilitation effect of 32 ischemic-hypoxic encephalopathy patients who were received rehabilitation therapy were retrospectively analyzed.
结论：缺血缺氧性脑病是引起新生儿惊厥的原因之一，窒息越重，EEG阳性率越高。
Conclusion: HIE is one of the main cause of neonatal convulsion. The more serious the asphyxia, the higher the positive rate of EEG.
方法回顾性分析64例应用二维超声诊断为新生儿缺血缺氧性脑病的临床及影像资料。
Methods 64 patients' clinical and image data were reviewed and analyzed with HIE diagnosed by ultrasound.
目的：探讨早期触摸和环境刺激对宫内缺血缺氧性脑损伤(HIBD)大鼠脑功能的影响。
Objective: To study the effect of the neonatal handling and enriched environmental stimulation on brain function of rats with hypoxic-ischemic brain damage (HIBD) in uterus.
前言： 目的：观察新生儿缺血缺氧性脑病（H IE）早期脑电地形图（B EAM）的变化及其意义。
Objective:To observe the change and significance of BEAM in the early period of newborn HIE.
目的研究氟桂利嗪(FNZ)、拉莫三嗪(ltg)及两药联合经孕鼠给药，对胎鼠宫内缺血缺氧性脑损害的保护作用。
Objective To investigate the neuroprotective effects of flunarizine (FNZ) and lamotrigine (LTG), and their combination on the hypoxic-ischemic brain damage in fetal rats.
肝组织均未发生显著变化。结论宫内窘迫时，脑组织首先出现自由基变化，为新生儿缺血缺氧性脑损伤的早期防治提供了理论依据。
Conclusion After severe intrauterine fetal distress, changes of free radicals occurred in brains firstly, which provides the proof of early treatment for neonatal hypoxia ischemia brain damage.
结果：57例患儿中，病毒性脑炎、高热、低钙伴惊厥者无一例并发症发生，全部治愈出院； 缺血缺氧性脑病伴惊厥的患儿病情得到及时控制，好转出院。
Result:In the 57 cases of children with infantile convulsion, the concomitant by viral encephalitis, fever and hypocalcaemia in patients without complications occurred, and all cured.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
结论：吸入性烧伤，严重休克，严重缺血、缺氧，复合脑外伤及麻醉意外等是出现呼吸、心跳骤停的主要原因；
Conclusion:The main cause resulting in cardiac arrest, respiratory arrest is inhalation burn , severe shock, short of oxygen, brain trauma.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
目的探讨新生儿缺氧缺血性脑病（HIE）临床与CT分度的相关性。
Objective To study the interrelation of clinic and CT graduation in the neonatal hypoxic-ischemic encephalopathy(HIE).
目的：观察新生大鼠缺氧缺血性脑损伤(HIBD)后海马区细胞增殖的情况，探讨脑组织内源性修复的可能机制。
Objective to observe the expression of proliferation in hippocampus following hypoxic-ischemic brain damage (HIBD) in neonatal rats and to explore the possible endogenous neurogenesis of brain.
结论：积极合理而有效的护理措施是新生儿缺氧缺血性脑病综合治疗的关键性环节。
Conclusion: It's the key point to nurse neonatal hypoxic ischemic encephalopathy effectively and properly for brain injury.
目的探讨新生儿中重度缺氧缺血性脑病(HIE)早期微量母乳喂养的可行性和疗效。
OBJECTIVE To investigate the feasibility and effect of early minimal enteral breast feeding (MEBF) in neonates with moderate and severe hypoxic-ischemic encephalopathy (HIE).
目的探讨内源性一氧化碳（CO）在新生大鼠缺氧缺血性脑损伤（HIBD）中的作用机制及锡原卟啉的保护作用。
Objective To study the role of endogenous carbon monoxide(CO) in newborn rats with hypoxic-ischemic brain damage (HIBD) and the protective effect of Sn-protoporphyrin (SnPP).
方法测定对诱发性心肌缺血、失血性冠状动脉主干结扎性心肌缺血、失血性休克、耐缺氧的影响。
METHOD to determine the effect of the preparations on induced myocardial ischemia, myocardial ischemia due to ligation of the main coronary artery, ischemic shock and hypoxia.
结论：新生大鼠缺氧缺血性脑损伤神经元退行性变形式多样。
Conclusion: This neuron degeneration of HIE in neonatal rats displayed a morphological diversity.
该文主要阐述细胞凋亡与新生儿缺氧缺血性脑损伤的关系，提出细胞凋亡机制在缺氧缺血性脑损伤中的重要性。
In this review, we summarized relationship between apoptosis and neonatal hypoxic-ischemic brain damage (HIBD) and importance of apoptosis mechanism in HIBD.
结论“反转征”是新生儿围产期窒息后重度缺氧缺血性脑病的少见征象，CT具有特征性表现。
Conclusion The cerebral ct "reversal sign" is a rare and characteristic feature of severe asphyxia in newborn.
结论“反转征”是新生儿围产期窒息后重度缺氧缺血性脑病的少见征象，CT具有特征性表现。
Conclusion The cerebral ct "reversal sign" is a rare and characteristic feature of severe asphyxia in newborn.
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