目的研究外源性单唾液酸四己糖神经节苷脂（GM1）对新生鼠缺氧缺血脑损伤（HIBD）的保护作用。
AIM To study the protective effects of monosialotetrahexosylganglioside (GM1) after hypoxic ischemic brain damage (HIBD) in neonatal rats.
目的探讨N-乙酰半胱氨酸（NAC）对缺氧缺血脑损伤（HIBD）大鼠脑细胞间黏附分子-1及细胞凋亡的影响。
Objective To explore the effect of N-acetylcysteine (NAC) on intercellular adhesion molecule-1(ICAM-1) expression and apoptosis in neonatal rats brain after hypoxic and ischemic brain damage (HIBD).
目的研究缺氧缺血新生猪凝血状态，以及部分凝血因子促凝活性的动态变化，探讨缺氧缺血性脑损伤（HIBD）时凝血功能变化。
Objective To investigate the changes of blood coagulation state and some prothrombin's function in newborn piglets at different time after hypoxic-ischemic brain damage (HIBD).
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
目的探讨超声诊断在新生儿缺氧缺血性脑病(HIE)各种脑损伤中的应用价值。
Objective To study the application of ultrasound diagnosis of various brain injury caused by hypoxic-ischemic encephalopathy (HIE).
目的探讨早期干预对缺氧缺血性脑损伤(HIBD)大鼠学习记忆能力及神经元凋亡的影响。
Objective to explore the influences of early interventions on learning, memory and neuron apoptosis in the brain of neonatal rats with hypoxic ischemic brain damage (HIBD).
目的利用氟桂利嗪(FNZ)研究钙离子拮抗剂对体外培养海马神经元缺血缺氧损伤的作用及其作用机制。
Objective To study the dual effects of calcium antagonists on ischemic hypoxic injury of hippocampal neurons cultured in vitro by using flunarizine (FNZ) and the mechanism of its action.
目的研究新生猪缺氧缺血脑损伤(HIBD)模型的制备。
Objective To develop an improved neonatal piglet model of hypoxic ischemic brain damage (HIBD).
通过亚低温对新生鼠缺氧缺血性脑损伤（HIBD）大脑皮质神经元一氧化氮合酶（NOS）及血糖水平影响的研究，探讨亚低温对缺氧缺血性脑损伤的保护作用机制。
Through investigating the effect of mild hypothermia on activity of nitric oxide synthase(NOS) in cortical neurons and glycemia levels of neonatal rats with hypoxic ischemic brain damage(HIBD).
目的观察缺氧缺血性脑损伤新生猪脑电生理的变化。
Objective To observe the electric physiologic changes in newborn piglets with HIBD.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
结果纳入缺氧缺血性脑损伤患儿148例，全部进入结果分析，无脱落。
RESULTS: All the 148 cases of hypoxia-ischemia brain injury were involved in the result analysis.
研究谷胱甘肽对缺氧缺血脑损伤引起的神经细胞凋亡的作用。
To study the effects of glutathione on hypoxic ischemic brain damage in neonatal rats hippocampal.
目的探讨鹿石合剂对缺氧缺血脑损伤新生大鼠保护作用的机制。
Objective To explore the protective effect of Lushi mixture on hypoxic-ischemic brain damage neonatal rats.
目的探索东莨菪碱对新生儿缺氧缺血性脑损伤（HIBD）的治疗作用。
Objective To evaluate treatment response of scopolamine for hypoxic-ischaemic brain damage(HIBD) in neonates.
目的研究新生动物缺氧缺血性脑损伤（HIBD）后脑细胞间粘附分子-1（ICAM- 1）在转录水平表达规律。
Objective To study the expression of ICAM 1 at gene transcription level in neonatal rats after hypoxic ischemic brain damage(HIBD).
目的探讨内源性一氧化碳（CO）在新生大鼠缺氧缺血性脑损伤（HIBD）中的作用机制及锡原卟啉的保护作用。
Objective To study the role of endogenous carbon monoxide(CO) in newborn rats with hypoxic-ischemic brain damage (HIBD) and the protective effect of Sn-protoporphyrin (SnPP).
目的探讨1,6 -二磷酸果糖(FDP)治疗新生儿缺氧缺血性脑病(HIE)合并心肌损伤的疗效。
Objective: to observe the clinical effect of fructose-1, 6-diphosphate (FDP) in neonate hypoxic ischemic encephalopathy (HIE) with myocardial injury.
目的了解新生鼠缺氧缺血性脑损伤(HIBD)时大脑皮质神经元特异性烯醇化酶(nse)和胶质纤维酸性蛋白(GFAP)的变化。
Objective To observe the change of neuron-specific enolase (NSE) and glial fibrillary acidic protein (GFAP) in neonatal rats' cerebral cortex with hypoxic-ischemic brain damage (HIBD).
结论：NOS抑制剂通过减轻缺血缺氧脑损伤时胎鼠脑神经细胞凋亡对其出生后的远期智能起保护作用。
Conclusion: NOS inhibitors play a obvious protective role in long-term intelligence of fetal rat after delivery through relieved neuron apoptosis during hypoxic ischemia encephalopathy.
结论：应用基因芯片技术筛选了与IEC缺血缺氧损伤密切相关的差异表达基因，为阐明这方面的机制提供了新的线索。
CONCLUSION: cDNA microarray can be used to screen diversified gene expression related to injury under ischemia and anoxia, which brings some new clues for studying the mechanism of IECs injury.
围产期窒息所致缺氧缺血性脑损伤(HIBD)是新生儿死亡和残疾的主要原因。
Hypoxia ischemic brain damage (HIBD), which resulted from neonatal asphyxia, is the main cause of death and disability in neonatal periods.
新生儿缺氧缺血性脑病(HIE)是指围产期缺氧窒息，导致脑的缺氧缺血性损伤。
Hypoxic ischemic (HIE) of newborn is the damage in brain because of perinatal hypoxia or asphyxiation.
背景：脑损伤后常引起继发性脑缺血、缺氧，使脑损害加重。
BACKGROUND: brain injury often causes secondary cerebral ischemia and hypoxia, which aggravate the brain damages.
黄芪用于治疗新生儿缺氧缺血性脑病能缩短疗程，促进新生儿行为神经功能的恢复，减轻脑损伤，调节细胞免疫功能。
In treating NHIE, Radix Astragali injection can shorten the treatment course, promote the recovery of NBNA, reduce brain damage and regulate cellular immune function.
目的：探讨早期触摸和环境刺激对宫内缺血缺氧性脑损伤(HIBD)大鼠脑功能的影响。
Objective: To study the effect of the neonatal handling and enriched environmental stimulation on brain function of rats with hypoxic-ischemic brain damage (HIBD) in uterus.
目的观察缺氧缺血性脑损伤（HIBD）新生大鼠白细胞介素-16（IL-16）表达的变化。
Objective To observe the variations in the expression levels of interleukin-16(IL-16) protein in neonatal rats with hypoxic-ischemic brain damage(HIBD).
近年来认识到脑缺血在缺血性损伤过程中除缺氧和能量代谢衰竭外，由缺血诱导的一系列瀑布样效应是导致缺血性神经元死亡的重要机制。
Recently, it has been found that a series cascade damages induced by ischemia is the important mechanisms of ischemic neuron death, in addition to anoxia and energy failure.
结论：脑活素可通过提高血钙、镁浓度以改善缺氧缺血性脑病损伤。
Conclusions: Cerebrolysin can decrease the encephalic injury by increasing the concentration of blood calcium and magnesium.
肾缺血后损伤显示，损伤不仅是血供中断后组织缺氧的结果，更有再灌注进程中引起炎症反应的激活过程。
Renal injury after ischemia appears to be a consequence of tissue hypoxia not only from interrupted blood supply but also from the process of reperfusion which leads to an active inflammatory process.
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