术后病理学检查发现，观察组癌细胞均有不同程度的变性、坏死，细胞间质水肿，纤维增生，炎性细胞浸润；
The post-operative pathological examination showed varying degrees of apomorphosis and necrosis of tumor cell, intercellular substance edema, fibrous hyperplasia and inflammatory cell invasion.
组镜下见肺灶性出血，间质、肺泡水肿，大量炎细胞浸润。
Local hemorrhage, interstitial edema, alveoli exudation and inflammatory cells were observed in lungs in group a.
与对照组相比较，各时间点的实验组大鼠肺组织炎症细胞渗出减少，间质水肿减轻。
In the study group, pulmonary architecture was preserved and infiltration of inflammatory cells and edema decreased.
间质结构松弛，血管上皮缺损及成纤维细胞水肿。
The interstitium was loose in texture, the endothelium of blood vessels was damaged and fibroblasts were edematous.
引起滋养层细胞肿胀和绒毛间质水肿。
Causes the trophoderm cell swelling and down mesenchymal dropsy.
第9至12周可见间质细胞增生、间质水肿、肺泡内出现粉红色泡沫样渗出物、部分肺组织呈现大片状实变区。
In 9 th to 12 th week, proliferation of interstitial cells, interstitial edema, red foamy alveolar exudates were observed.
肾间质水肿伴炎症细胞浸润，部分小管上皮细胞变性、坏死并见管型，死亡率2 0 %。
The histologic changes of renal tissue were interstitial edema with inflammatory cellular infiltration, denaturalization and necrosis in some tubular epithelia. LPS caused 20% rats death.
有时这种支持细胞间质表现出显著的硬化或水肿。
Sometimes the supporting stroma shows significant sclerosis or edema.
有时这种支持细胞间质表现出显著的硬化或水肿。
Sometimes the supporting stroma shows significant sclerosis or edema.
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