这种疗法昂贵的部分原因在于,它不是一种实验室研制的药物,而是针对个体需要而采取的特定疗法,需要用到每位患者的细胞,以及大多数前列腺癌细胞所具有的蛋白质。
Part of why it costs so much is that it's not a pill cranked out in a lab, but a treatment that is individually prepared, using each patient's cells and a protein found on most prostate cancer cells.
他说,这些药物对于实验室内的人体细胞很有效果,但是在正式成为治疗手段之前还需进行药物毒理和临床实验,而这个过程大概需要五年或更长时间。
He said that the drugs have been effective on human cells in the laboratory but that toxicology and clinical trials would be needed before it could be adopted as a treatment.
这种药物在实验室中被发现可以抑制早衰细胞的变异,并在实验鼠身上产生了预期效果。
These were found to reverse an abnormality in progeria cells in the laboratory and also prevented symptoms developing in progeria mice.
在实验室中,也可以直接将药物作用与带病细胞上以检验药性。
Drugs that may show promise in slowing or stopping the condition can be tested by adding them directly to the disease-carrying brain cells in the lab.
为了论证这点,Baker和van Deursen繁殖了一组具有快速衰老基因的实验鼠,在这个过程中,对实验鼠施以药物刺激,从而清除脂肪组织、肌肉和眼睛中含有P16的细胞。
To accomplish this, Baker and van Deursen designed a fast-aging mouse strain that would, upon receiving a drug trigger, expel p16-producing cells from fatty tissues, muscles and eyes.
根据派普和同事在《细胞》(Cell)期刊上发表的报告,因为与Medivation和辉瑞制药的实验性阿斯海默症治疗药物Dimebon 成份相似,这种化合物有可能提供更多改善该药药效的方法。
The compound is similar to Medivation Inc and Pfizer Inc's experimental Alzheimer's drug, Dimebon, and may provide ways to improve its effects, Pieper and colleagues reported in the journal Cell.
华盛顿报道,研究人员发现一种能够帮助大脑生成新细胞的药物,这项研究可能有利于改进治疗阿兹·海默症(即老年痴呆症)的实验性药物。
WASHINGTON - Researchers have found a drug that can help the brain grow new cells and said their study may lead to ways to improve experimental Alzheimer's drugs.
细胞培养实验也表明有KEAP1突变的癌细胞对化疗药物的抵抗性强于正常肺细胞。
Their cell culture tests also show that cancer cells with KEAP1 mutations are more resistant to chemotherapy drugs than normal lung cells.
研究人员针对人肺癌肿瘤(A549细胞)在实验室小鼠临床实验中测试了飞毯药物输送技术。
The researchers tested the flying carpet drug delivery technique in preclinical trials against human lung cancer tumors (cell line A549) in laboratory mice.
培养的第2~3代细胞适用于缺氧实验和药物作用机制的研究。
Cultured pulmonary trunk wall cells at the 2nd to 3rd passage were applicable to investigations of hypoxic experiments and efficiency of drugs.
我们的研究使的研究人肝发育的潜在分子机制变得容易,并且形成肝细胞移植和药物实验的基础。
Our studies should facilitate searching the molecular mechanisms underlying human liver development, and form the basis for hepatocyte transplantation and drug tests.
在AEP基因敲除的小鼠实验中,该药物及人为中风所导致的DNA损伤与脑细胞死亡均比非基因敲除小鼠程度减轻。
In mice genetically engineered to lack AEP, both the drug and an artificial stroke resulted in reduced DNA damage and less brain cell death than in regular mice.
磁场结合抗肿瘤药物作用于肿瘤细胞后能有效抑制肿瘤生长,这种观点己被近年来的许多实验所证实。
The tumor growth can be significantly repressed by combining magnetic field with anti-tumor drugs, which has been proved by a series of experiments.
在动物实验研究中,治疗癫痫的药物在未成年的大脑中可引起细胞凋亡。
In animal studies, certain seizure medications can cause cell death in immature brains.
采用MTT实验研究细胞对铂类药物耐药性的影响。
MTT assay was used to study the effect on cytotoxicity and drug resistance in the cell lines.
实验表明,该化合物对多种肿瘤细胞具有良好的抑制活性,可用于制备抗肿瘤药物。
Experiments show that the compound has good activity for inhibiting various tumor cells and can be used for the preparation of anti-tumor drugs.
本实验用化学药物对家兔胚胎细胞周期进行调控,以建立细胞周期同期化的方法。
The objective of this experiment was to control the rabbit embryonic cell cycle with chemicals to give programs for the cycle synchronization.
最近实验室研究已经发现少量药物已经影响到了人类胚胎肾细胞、人类血细胞和人类乳腺癌细胞。
Recent laboratory research has found that small amounts of medication have affected human embryonic kidney cells, human blood cells and human breast cancer cells.
本文应用HAG封闭小鼠mps吞噬功能,用碳廓清实验和组织免疫荧光染色筛选了增强单核-巨噬细胞系统吞噬功能的药物。
The drugs that can strengthen phagocyte function of MPS was selected by applying hag inhibiting phagocyte function of MPS, using the test of clearance of carbon and immunofluorescence staining.
这种能扎根于被感染的细胞,使它们自我凋亡的药物已经在实验室里制造出来。
A drug that homes in on infected cells and makes them self-destruct has been created in the laboratory.
实验结果提示,淫羊藿为一潜在抗肿瘤药物,其诱导细胞程序死亡机制有待进一步探讨。
These results suggested that EGM is a potential antitumor drug and there is much to be done to study its mechanism to induce apoptosis.
“直到目前,大部分药物实验只在标准的肿瘤细胞系中测试,而这些细胞系很少含有肿瘤干细胞,”Ince解释到。
"Until now, most of the drug testing had been done on standard tumor cell lines that only have very few tumor stem cells in them," Ince explained.
目的:培养建立中国人种前列腺癌细胞系,为了解中国人种前列腺癌细胞生长特性、进一步开展前列腺癌基因诊断和药物筛选建立方便的实验手段和模型打下基础。
Objective: To culture and establish Chinese ethnic prostate cancer cell line, investigate its growth characterization and explore experimental methods and models of gene diagnosis and trial of drugs.
戴稼禾,等?黄耆药物对人体红细胞变形能力作用的实验研究?贵州医药1987;11(1):23。
Dai Jiahe, et al, Experimental study of effects of Radix Astragali on human erythrocyte deformation , Guizhou Medicine 1987; 11(1):23.
研究表明灵菌红素抑制细胞的增殖,与抑制S期细胞的DNA复制及调控细胞的增殖周期相关。 同时,细胞凋亡的产生与实验药物呈正相关。
The inhibitive effect of prodigiosin on multiplication of cancer cells was related to the inhibiting of DNA replication in S stage and regulating of cell cycle.
对40例肺癌、42例大肠癌患者作了白细胞对抗癌药物敏感性实验,同时以癌细胞药敏实验作对照。
The sensitivities of cancer cells and peripheral blood leukocytes(PBL) to anti cancer drugs among 40 cases of lung cancer and 42 cases of large intestinal cancer were tested by MTT method.
耶鲁大学的研究人员发现,这个缺陷引起实验室的脑肿瘤细胞对治疗一些卵巢癌女性的药物变得高度的敏感。
Researchers at Yale found that this weakness caused brain tumour cells in the lab to become highly sensitive to a drug that's used to treat some women with ovarian cancer.
我们经过统计与实验有力的发现,药物缓慢作用于T淋巴细胞而提升CD4的机理,才是根本。
We have a strong statistical and experimental findings, the drug slowly in the role of t lymphocytes and enhance the mechanism of CD4, is fundamental.
实验结果表明该药物载体具有很好的生物兼容性和化学稳定性,并在细胞中依然保持了很高的SERS活性,可进一步用于细胞内药物示踪检测。
The experimental results show that the drug carrier offers biocompatibility, stability, and high SERS activity, holding the potential for realizing the intracellular drug tracing.
实验结果表明该药物载体具有很好的生物兼容性和化学稳定性,并在细胞中依然保持了很高的SERS活性,可进一步用于细胞内药物示踪检测。
The experimental results show that the drug carrier offers biocompatibility, stability, and high SERS activity, holding the potential for realizing the intracellular drug tracing.
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