胰岛素样生长因子系统在调节细胞增殖、分化与抑制凋亡中发挥重要作用。
Insulin-like growth factor (IGF) system plays an important role in regulating cell proliferation and differentiation, and inhibiting apoptosis.
目的观察巨噬细胞移动抑制因子在大肠癌中的表达及其与细胞凋亡的关系。
Objective to observe the expression of macrophage migration inhibition factor (MIF) in colorectal carcinoma tissue and its correlation with cell apoptosis.
目的观察血管内皮生长因子(VEGF)抗体对大鼠c6胶质瘤细胞凋亡的影响及对肿瘤生长的抑制作用。
Objective to observe the affect of vascular endothelial growth factor (VEGF) antibody on the cell apoptosis and growth of rat C6 glioma.
目的研究组织金属蛋白酶抑制因子3（TIMP-3）重组蛋白对MC3T3-E1成骨细胞凋亡的影响。
Objective To investigate the action of recombinant protein of tissue inhibitor of metalloproteinase-3(TIMP-3)on apoptosis of MC3T3-E1 osteoblasts.
目的研究角质形成细胞生长因子(KGF)及其受体(KGFR)反义寡核苷酸对KGF介导的细胞周期和凋亡变化的抑制作用。
Objective to investigate the effects of keratinocyte growth factor (KGF) and KGF receptor (KGFR) antisense oligonucleotide (ASODN) on cell cycle and apoptosis of HaCat cells.
这和最近的研究是一致的，该研究表明卵母细胞成熟抑制因子能诱导用TNF处理的人类神经多肽的凋亡而不从线粒体中释放出来（7）。
This is in accord with a recent study that shows Omi can induce apoptosis in human neutrophils treated with TNF- without being released from the mitochondria (7).
该试验清楚地表明了卵母细胞成熟抑制因子单独决定HAX - 1的酶切，在本实验使用的条件下对于细胞凋亡是必须的。
This experiment clearly shows that Omi is solely responsible for HAX-1 cleavage, which is essential for apoptosis under the conditions used in these experiments.
这暗示着卵母细胞成熟抑制因子靠酶切hax -1蛋白来启动线粒体中的细胞凋亡。
This suggests that Omi can initiate apoptosis in the mitochondria by cleaving HAX-1 protein.
近些年来，研究发现IGF - 1不仅具有胰岛素类似的功能以及介导生长激素的作用，还是多种类型细胞凋亡的一个重要抑制因子。
Recent studies implicate IGF-1 not only ACTS as insulin and mediates the growth hormone action, but also can inhibit apoptosis and stimulate cell proliferation and differentiation.
目的：探讨肿瘤坏死因子相关细胞凋亡诱导配体(TRAIL)对人卵巢癌细胞株体外生长抑制作用。
Objective: the study is to find out the inhibition action of TNF-related apoptosis-inducing ligand or APO-2ligand (TRAIL) on ovarian carcinoma cell line cultured in vitro.
目的：探讨肿瘤坏死因子相关诱导凋亡配体(trail)蛋白和顺铂协同抑制横纹肌肉瘤细胞生长和诱导凋亡作用及其机制。
AIM: to study the synergistic induction of apoptosis by TRAIL and cisplatin in rhabdomyosarcoma cells and investigate the role of mitochondrial membrane potential and caspase-3 in this process.
目的：探讨肿瘤坏死因子相关诱导凋亡配体(trail)蛋白和顺铂协同抑制横纹肌肉瘤细胞生长和诱导凋亡作用及其机制。
AIM: to study the synergistic induction of apoptosis by TRAIL and cisplatin in rhabdomyosarcoma cells and investigate the role of mitochondrial membrane potential and caspase-3 in this process.
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