方法肝细胞和肝窦内皮细胞取自小鼠肝脏。
Methods Hepatocytes and sinusoidal endothelial (se) cells were harvested from mouse liver.
结果：边缘窦内皮细胞能吞噬、消化、处理炭粒。
Results: the marginal sinus endothelial. cells possessed the function of phagocytosis, digestion and removing of carbon particles.
淋巴细胞、成纤维细胞及血窦内皮细胞染色阳性。
Lymphocyte, fibroblast and sinusoid endotheliocyte were stained positively.
结果VEGF在肝组织表达有胞浆型、膜窦型及窦内皮细胞型；
Results The expression types of VEGF in liver tissue were plasma type, sinusoid membrane type and sinusoid endothelial cells type.
结论肝窦内皮细胞是脓毒症时肝组织中细胞因子的重要来源之一。
Conclusions Liver sinusoidal endothelial cell is an important source of cytokine production in mice with sepsis.
目的：为了进一步认识边缘窦内皮细胞在血脾屏障中的地位和作用。
Objective: To understand the function of splenic marginal sinus endothelial cells in blood spleen barrier.
目的研究肝窦内皮细胞损伤在二甲基亚硝胺大鼠肝纤维化形成中的作用。
Objective To study the role of hepatic sinusoidal endothelium injury during hepatic fibrogenesis induced by dimethylnitrosamine (DMN) in rats.
与对照组比较，实验组大鼠肝细胞和肝窦内皮细胞变性和坏死程度较轻。
Compared with the control group, the degenerative and necrotic degree of hepatocyte and sinusoidal endothelial cells was lighter in the experiment group.
本文对肝窦内皮细胞在其中的重要作用及减轻这一损伤的研究进展简要综述。
So, this article reviews the importance of endothelial cells in hepatic sinus and study progresses on the question tha…
番鸭呼肠孤病毒的主要靶细胞：肝脏内的肝细胞、肝血窦内皮细胞、枯否氏细胞；
The target cells of MDRV were as follows, Liver cells, Endothelial of blood sinus, Kupffer's cells in Liver;
病理基础是预处理毒性引起肝脏内肝小叶3区内的血窦内皮细胞以及肝细胞损害所致。
The basis of pathology of vod is thought resulting from injury of hepatocytes and endodermis cells surrounding the central veins in zone3of the liver acinus by regimen-related toxicity.
目的：探讨粒细胞和粘附分子在肝窦内皮细胞（LSEC）缺血再灌注损伤中的作用。
Objective:To investigate the role of granulocyte and adhesion molecules in the ischemia reperfusion injury of human liver sinusoidal endothelial cells(LSEC).
目的建立大鼠肝窦内皮细胞(sec)的原代分离、培养方法，并研究其生物学特性。
Objective To set up a method for the primary isolation and cultivation of rat sinusoidal endothelial cells (SEC), and study their biological characteristics.
结论D MN引起大鼠肝窦内皮细胞损伤及其表型改变可能是其诱导肝纤维化重要的始动机制之一。
Conclusions the damage and phenotypic alteration of the hepatic sinusoidal endothelium may be a vital issue triggering the liver fibrosis induced by DMN.
VEGF主要由肝细胞分泌，其次在肝窦内皮细胞、贮脂细胞、成肌纤维细胞及单型核细胞也示阳性。
VEGF was mainly secreted by hepatocytes, and was also positively expressed in liver sinusoid endothelial cells, fat-storing cells, myofibroblast and monomorphic nucleus cells.
应用一氧化氮和基质蛋白酶的抑制剂明显降低肝窦内皮细胞的损伤，表明一氧化氮和基质蛋白酶在缺血-再灌注损伤中是重要的介导物。
The inhibitor for NO or for MMPs significantly reduced the LSEC IRI, suggesting that NO and MMPs are important mediators in the LSEC IRI.
肝星状细胞、成纤维细胞和窦状隙内皮细胞是肝内ctgf的重要来源。
It can be concluded that CTGF was mainly produced in hepatic stellate cells, sinusoidal endothelial cells and fibroblasts.
目的研究门静脉高压症病人肝组织内血管及肝窦壁内皮细胞的凋亡及其对肝微循环的影响。
Objective To investigate the intrahepatic vascular and hepatic sinusoidal endothelial cell (SEC) apoptosis and its influence on hepatic microcirculation in patients with portal hypertension.
海绵状血管畸形由窦状扩张的血窦组成，窦壁由内皮细胞，基膜物质和周细胞组成。
There were endothelial cells , basal membrane like materials and pericytes in the walls of these blood sinus.
结论：肝窦状内皮细胞在体外培养条件下自发永生化。
Conclusions: in vitro sinusoid endothelial cells could spontaneously immortalize. Some biological characteristics of immortalized cells changed.
作为血窦屏障中驻留的前骨细胞和周围内皮细胞，骨前体统一了成骨细胞壁龛和窦状隙壁龛两个概念。
As pre-osteoblastic, peri-endothelial cells residing at the sinusoid wall, skeletal progenitors reconcile the notions of "osteoblastic" and "sinusoidal" niches with one another.
CTGF的表达主要集中于汇管区和纤维化区，肝星状细胞、成纤维细胞和窦状隙内皮细胞染色常呈阳性。
CTGF was mainly expressed in portal tracts and fibrotic areas. Positive staining was observed in hepatic stellate cells, sinusoidal endothelial cells and fibroblasts.
CTGF的表达主要集中于汇管区和纤维化区，肝星状细胞、成纤维细胞和窦状隙内皮细胞染色常呈阳性。
CTGF was mainly expressed in portal tracts and fibrotic areas. Positive staining was observed in hepatic stellate cells, sinusoidal endothelial cells and fibroblasts.
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