• 炎性细胞因子介导白细胞活化就是SIRS直接结果

    The activation of leukocyte mediated by the cytokinesis the direct result of SIRS.

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  • 再灌注期间上皮细胞功能障碍内源激活白细胞聚集活化,都会导致自由基产生

    During the reperfusion period, endothelial dysfunction, activation of endogenous enzymes, leucocyte recruitment and activation all lead to the generation of oxygen -derived free radical.

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  • 结果活化白细胞克隆到IDO基因编码区全长,构建了IDOEGFP融合蛋白表达载体

    Results The full-length coding sequence of IDO was cloned from activated human leukocytes and the expression vector for IDO-EGFP fusion protein was constructed.

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  • WCD降低白细胞活化有关

    The lower WCD were related to WBC activation.

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  • 人类白细胞抗原d R位点(HLA - DR)表达作为晚期(刺激2 - 4)T淋巴细胞活化的指标。

    The expression of human leukocyte antigen-DR (HLA-DR) is taking as a maker for later phase (2-4 days after stimulate) of lymphocyte activation.

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  • 目的研究白细胞介素- 2 (IL - 2)白细胞介素- 18 (IL - 18)诱导自然杀伤(NK)细胞活化和抗骨髓瘤细胞活性协同作用

    Objective To explore potential synergetic effects of interleukin-2 (IL-2) with IL-18 on induction of natural killer (NK) cell activity against myeloma cells in vitro.

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  • 目的研究白细胞介素6IL 6 )依赖型骨髓瘤细胞系XG7中IL 6信号转导途径活化生物学效应之间关系

    Objective To investigate the relationship between IL 6 signal transduction and its biological function on a human IL 6 dependent myeloma cell line XG 7.

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  • 结果C5暴露组织蛋白酶G弹性酶时产生C5片段,而C5片段能使活化白细胞释放MPO和乳铁传递蛋白。

    Results C5 exposed to cathepsin G and elastase generated C5 fragments. C5 fragments stimulated primed neutrophil to release myeloperoxidase(MPO) and lactoferrin.

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  • 目的:探讨皮质激素对于活化rbl-2h3(简称rbl)表达白细胞介素5 (IL - 5)影响

    Objective To investigate the influences of glucocorticoids on interleukin - 5 (IL - 5) expression in RBL - 2h3 (RBL).

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  • 目的研究细胞白细胞介素- 10 (IL - 10)原代状细胞(hsc)增殖活化及凋亡影响,并探讨其可能机制。

    Aim: to investigate the effects of hepatocyte line and interleukin-10 (IL-10) on the proliferation, activation and apoptosis of primary hepatic stellate cells (HSC).

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  • 目的研究细胞白细胞介素- 10 (IL - 10)原代状细胞(hsc)增殖活化及凋亡影响,并探讨其可能机制。

    Aim: to investigate the effects of hepatocyte line and interleukin-10 (IL-10) on the proliferation, activation and apoptosis of primary hepatic stellate cells (HSC).

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