你其中的一些有来新鲜的从狭窄的细胞。
通过电子显微镜可明确地观察到当白细胞通过内皮细胞之间狭窄、蜿蜒的通道时，显示出难以置信的可塑性。
Electron microscopy has elegantly shown the incredible plasticity of the relatively bulky white cells as they crawl through the narrow, serpentine pathways between endothelial cells.
而平滑肌细胞凋亡与动脉粥样硬化和血管成形术后再狭窄密切相关。
The apoptosis of smooth muscle cells was related to the atherosclerosis and restenosis of vessels.
通过喉狭窄瘢痕组织的病理学研究，了解炎症细胞在狭窄瘢痕组织表达及意义。
To explore the significance of the expression of inflammation cell in the laryngeal stricture scar by the study of the pathological features of laryngeal stenosis scar.
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
结果先天性巨结肠狭窄部肠壁肌间神经无神经细胞，可见增多的直径增粗无髓鞘纤维，呈波浪状弯曲。
Results The ganglion cell wasn't found in myenteric nerve plexus of the constriction section. The diameter and the number of the nerve fibers were increased; Schwann's cells were increased.
目的：探讨碱性成纤维细胞生长因子对基质金属蛋白酶表达的影响及其在球囊术后再狭窄中的作用。
Objective: To investigate the effect of basic fibroblast growth factor on matrix metalloproteinases and restenosis after PTCA .
它充满并推向细胞的顶部，迫使核移向狭窄的基部。
It fills and expands the apical portion of the cell and forces the nucleus into the slender basal portion.
研究者还认为，它有助于红细胞保持弹性，使他们能够很容易进入狭窄制约的血管。
The researchers also believe that it helps red blood cells to stay flexible so they can easily fit into the narrow constraints of the blood vessels.
血管内放射导致细胞死亡的主要形式为凋亡，电离辐射预防再狭窄的主要机制是平滑肌细脑的增殖抑制。
Apoptosis is a major style of cell death induced by endovascular radiation. The inhibition of SMCs proliferation induced by radiation is a main mechanism for prevention of restenosis.
研究喉狭窄瘢痕组织的病理变化，了解细胞外基质的变化及意义。
To explore the significance of the change of extracellular matrix in the laryngeal stenosis scar by the study of the pathological features of laryngeal stenosis scar.
P 27蛋白的高表达可显著抑制血管平滑肌细胞增殖，有望成为治疗PT CA后再狭窄的新手段。
P27Kipl restrains the proliferation of VSMC obviously and is prospective to become a new means to cure restenosis after PTCA.
称为再狭窄的这个过程一直被描述为组织的超强愈合能力，包括平滑肌细胞。
This process, known as restenosis, has been described as "over exuberant" tissue healing and involves the smooth muscle cells.
组织学观察显示，实验组动物气管狭窄部位成纤维细胞增生，胶原纤维增厚。
Proliferation of fibroblasts and thickening of collagen fibers in areas of tracheal stenosis were observed by microscopic observations.
背景角膜再生和修复由角膜缘干细胞介导，角膜缘即角膜和球结膜间的狭窄区。
Background Corneal renewal and repair are mediated by stem cells of the limbus, the narrow zone between the cornea and the bulbar conjunctiva.
研究表明ROCK与血管再狭窄、心肌损伤、心肌细胞凋亡及心力衰竭等有关联。
It has been shown that ROCK is associated with restenosis, myocardial damage, myocardial apoptosis, and heart failure.
当血管损伤后，局部产生的细胞因子引起炎症反应是血管再狭窄的主要原因。
The vascular restenosis is attributable to a local inflammation after vascular injury, which results in the production of cytokines.
PTCA后白细胞的激活可能触发一系列病理生理联级反应，释放某些炎症介质，促进血管平滑肌增殖导致再狭窄；
Activation of leukocytes after PTCA can trigger a serial cascade of pathophysiologic reaction, releasing some inflammation intermediates lead to restenosis.
白细胞黏附素还可减少白细胞的募集并减少在大鼠中的损伤后动脉狭窄。
Leukadherins also decreased leukocyte recruitment and reduced arterial narrowing after injury in rats.
结果39例胆道恶性狭窄病例中胆汁细胞学检查阳性26例，敏感性66.7%，血清CA19-9检测阳性28例，敏感性71.8%；
Results Malignant cells were found in bile from 26 of 39 malignant obstruction (66.7%), and serum CA19-9 was positive in 28 (71.8%).
结果39例胆道恶性狭窄病例中胆汁细胞学检查阳性26例，敏感性66.7%，血清CA19-9检测阳性28例，敏感性71.8%；
Results Malignant cells were found in bile from 26 of 39 malignant obstruction (66.7%), and serum CA19-9 was positive in 28 (71.8%).
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