结论：同型半胱氨酸加重离体帕金森病模型动物的多巴胺能神经元损伤，其机制可能与氧化应激反应有关。
CONCLUSION: Homocysteine can aggravate the injury of dopaminergic neurons in animal models of Parkinson disease, and the mechanism may be associated with the anti-oxidative stress.
结论：同型半胱氨酸加重离体帕金森病模型动物的多巴胺能神经元损伤，其机制可能与氧化应激反应有关。
CONCLUSION: Homocysteine can aggravate the injury of dopaminergic neurons in animal models of Parkinson disease, and the mechanism may be associated with the anti-oxidative stress.
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