脂质过氧化产物丙二醛可引起溶血。
并检测血糖、糖基化血清蛋白和血清脂过氧化产物丙二醛浓度以反映代谢控制。
Serum concentrations of glucose, glycosylated serum protein, and malondialdehyde, derived from lipid peroxidation were also examined to estimate metabolic control.
结果发现，6-BA预处理抑制了膜透性的增大；过氧化产物丙二醛（MDA）的产生得到抑制；
The results showed that 6-BA suppressed the membrane permeability and delayed peroxidation of membrane lipids, so that the generation of MDA was inhibited.
方法测定6 8例老年性耳聋病人和5 0例健康老年人的血浆超氧化物歧化酶（SOD）和脂质过氧化产物丙二醛（MDA）的水平及血液红细胞变形性。
Methods The Red Cell Deformability (RCD)and the levels of the blood plasma SOD and MDA in 68 patients with presbycusis and in 50 healthy old people were measured.
丙二醛(MDA)是脂质过氧化产物的稳定代谢物。
Molondialdehyde (MDA) was the stable metabolite of lipid peroxidation products.
丙二醛(MDA)是膜脂过氧化的终产物，其含量可用来反映植物对逆境条件的反应强弱。
Malondialdehyde (MDA) is the final product of lipid peroxidation, and its content can reflect stress tolerance of plants.
叶片内脂质过氧化作用产物丙二醛（MDA）的产生减缓，MDA含量小且变化的波动较小一直保持在较低水平。
Membrane lipid per- oxidation production MDA come into being tardily, the contents of MDA low and change in the parochial bound.
叶片内脂质过氧化作用产物丙二醛（MDA）的产生减缓，MDA含量小且变化的波动较小一直保持在较低水平。
Membrane lipid per- oxidation production MDA come into being tardily, the contents of MDA low and change in the parochial bound.
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