结论支气管平滑肌mlck表达增加可能与哮喘发病有关。
Conclusion The expression increase of MLCK in bronchial smooth muscle may be correlated with asthmatic pathogenesis.
NO具有扩张血管平滑肌、松弛支气管平滑肌及降低肺动脉压、消除支气管痉挛的作用。
NO can lower the pressure of pulmonary artery and relieve the bronchospasm through dilating the smooth muscle of blood vessel and relaxing the smooth muscle of bronchus.
结论：MCT性PH时TERT在肺血管平滑肌、支气管平滑肌的过度表达在MCT性PH的发病机制中可能具有重要意义。
CONCLUSION: Over expression of TERT in pulmonary vascular smooth muscle cells and bronchial smooth muscle cells is related to MCT-induced PH.
在使用时通过该公司生产的Alair支气管热传导系统，将热能精确的传递到气管的特定部位，从而减少气管平滑肌过度反应。
In use, the company's Alair Bronchial Thermoplasty System delivers thermal energy to the airway wall in a precisely controlled manner to reduce excessive airway smooth muscle.
呼吸道平滑肌可见于气管至末梢细支气管中，它被认为是调节支气管运动张力的重要组织。
Airway smooth muscle (ASM), existing in the trachea and in the bronchial tree up to the terminal bronchioles, serves as an important structural effector tissue to regulate bronchomotor tone.
呼吸道平滑肌可见于气管至末梢细支气管中，它被认为是调节支气管运动张力的重要组织。
Airway smooth muscle (ASM), existing in the trachea and in the bronchial tree up to the terminal bronchioles, serves as an important structural effector tissue to regulate bronchomotor tone.
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