咪达普利抑制心房纤维化可能还与影响CTGF途径有关。
Imidapril inhibit atrial fibrosis maybe also related to affect the CTGF way.
贝那普利及缬沙坦可有效减轻心房纤维化和心肌缝隙连接蛋白的重构。
Benazepril and valsartan can decreased the degree of atrial fibrosis and remodeling of Gap junction connexin effectively.
高血压患者有较高心律失常的发生率，房性心律失常可能与左房扩大或心房纤维化有关。
Hypertensive patients have a higher incidence of atrial arrhythmia which might be related to atrial enlargement or fibrosis.
结论腹主动脉结扎大鼠心房纤维化程度增加，P波时限增宽，IACT延长，心房有效不应期没有改变。
Conclusion The extent of atrial fibro sis was increased in abdominal aorta banded rats. Although longer P wave duration and IACT are present, there is no change in atrial effective refractory period.
心房肌的纤维化与房颤的发生及维持有关。
Fibrosis of atrial myocytes may be involved in the occurrence and maintenance of AF.
目的探讨心房颤动心肌纤维化的发生机制。
Objective To investigate the mechanisms of the cardiac fibrosis during atrial fibrillation.
HE染色发现心房颤动患者的心房有肌溶解、心肌肥厚及广泛的间质纤维化改变。
In he stain, myocardial hypertrophy, severe myolysis and extensive fibrosis in the interstitial space of the atria were found in AF group.
充血性心力衰竭所致的心房间质纤维化，通过影响心房传导来促进心律失常的发生。
Congestie heart failure produces atrial interstitial fibrosis, which promotes arrhythmogenesis by interfering with atrial conduction properties.
充血性心力衰竭所致的心房间质纤维化，通过影响心房传导来促进心律失常的发生。
Congestie heart failure produces atrial interstitial fibrosis, which promotes arrhythmogenesis by interfering with atrial conduction properties.
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