目的探讨尿毒症患者左室重构的相关因素。
Objective To realize the effect of related factors on left ventricular structure changes of patients with uremia.
目的探讨高血压患者运动血压与左室重构的关系。
Objective To explore the relationship between blood pressure (BP) response to exercise and left ventricular remodeling in essential hypertension (EH).
防止左室重构进展是治疗急性心肌梗死(MI)的关键点。
Preventing progressive left ventricular remodeling is a key therapeutic objective after acute myocardial infarction (MI).
结论氯沙坦、培多普利可以减轻左室重构，改善心功能障碍。
CONCLUSION Losartan and Perindopril could improve the left ventricular remodeling and heart functions in AMI rats.
结论联合应用远端保护装置，可更加有效阻止左室重构的进程。
Conclusion Left ventricle remodeling was prevented effectively by using distal protection devices.
目的研究血压平逆转二肾一夹(2k1c)肾性高血压大鼠左室重构的作用及其机理。
Objective to investigate the reverse effect of Xueyaping on left ventricle remodeling resulting from renal hypertension induced by 2k1c in rats and to explore the mechanism of Xueyaping.
目的探讨复方中药制剂血压平逆转二肾一夹（2K1C）肾性高血压大鼠左室重构的作用及机制。
Objective To explore the reverse effect and its mechanism of Xueyaping on left ventricle remodeling resulting from renal hypertension induced by 2K1C in rats.
因此，研究心梗后左室重构的病理机制和探索合理的治疗措施是近年来冠心病研究的热点问题之一。
Therefore, the pathophysiologic process of left ventricular remodeling and to find better treatment have been better appreciated recently.
背景：压力负荷可以导致肾素血管紧张素醛固酮系统激活，进而导致左室重构，卡托普利对这种重构具有逆转作用。
BACKGROUND: Pressure overload can lead to the activation of renin-angiotensin-aldosterone system, and then left ventricular remodeling occurs. Captopril can reverse this progress.
目的评价经皮冠状动脉介入治疗(pci)对伴左心室收缩功能不全的冠状动脉慢性完全闭塞(CTO)患者左室重构及心功能的影响。
Objective to evaluate the effect of percutaneous coronary intervention (PCI) on left ventricular systolic function in patients with chronic total occlusion (CTO) and left ventricular dysfunction.
结论缬沙坦能改善向心性重构型原发性高血压患者左室整体舒张功能。
Conclusion Valsartan can improve the global left ventricular diastolic function in essential hypertension patients with left ventricular conventric remodeling.
但是，对轻中度收缩性HF患者，螺内酯联合血管紧张素ii受体拮抗剂对左室(LV)逆重构的价值目前还不清楚。
However, the value of combining spironolactone with an angiotensin II receptor blocker on LV reverse remodeling in mild-to-moderate systolic HF is unclear.
结果显示，横管重构在代偿性肥厚阶段即已发生，表现为左室部分细胞中离散性的局部区域横管缺失；
We found that t-tubule remodeling starts at the compensated hypertrophy stage featuring ast-tubule loss in discrete local regions of a small percentage of myocytes in the left ventricle.
结果显示，横管重构在代偿性肥厚阶段即已发生，表现为左室部分细胞中离散性的局部区域横管缺失；
We found that t-tubule remodeling starts at the compensated hypertrophy stage featuring ast-tubule loss in discrete local regions of a small percentage of myocytes in the left ventricle.
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