背景：压力负荷可以导致肾素血管紧张素醛固酮系统激活，进而导致左室重构，卡托普利对这种重构具有逆转作用。
BACKGROUND: Pressure overload can lead to the activation of renin-angiotensin-aldosterone system, and then left ventricular remodeling occurs. Captopril can reverse this progress.
在病人的肺水肿，静脉电容增加，从而减少左室充盈的压力。
In patients with pulmonary oedema, venous capacitance is increased, thereby decreasing left ventricular filling pressure.
跨室间隔压力差与左室舒张末偏心指数呈显著负相关。
The diastolic transseptal pressure gradient correlated inversely with the eccentric index.
跨室间隔压力差与左室舒张末偏心指数呈显著负相关。
The diastolic transseptal pressure gradient correlated inversely with the eccentric index.
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