方法清醒大鼠尾动脉无创血压测定法。
Methods: Blood pressures were measured indirectly by tail arteries in awake rats.
体和尾由胰背动脉、胰支、胰大动脉和胰尾动脉供血。
The body and tail were supplied by dorsal pancreatic artery, branches of pancreas, caudal pancreas artery and artery...
方法以SD大鼠为研究对象，尾动脉插管灌注，右颈静脉插管引流，建立常温大鼠体外循环模型。
Methods the tail artery serving as the arterial inflow and the right jugular vein as the venous drainage for the CPB circuit, we established the normothermic CPB model in SD rats.
实验过程中每周测量全部大鼠血压一次，采用尾动脉容积测压法观察对清醒SHR血压产生的效应。
Experiment process in every week measure all big rat blood pressure once, adoption tail artery the capacity measure to press a method observation lightness awake SHR blood pressure creation of effect.
方法术中结扎犬胰十二指肠前、后动脉、胰尾动脉。观察缺血后动物的生存状况及胰腺、十二指肠病理改变。
Mothods Ligated the anterior, posterior and rear pancreaticoduodenal artery, observed the state of animals survival and the pathology change of dog's pancreas and duodenum after ischemic operation.
前言： 目的观察清热解毒法抗SHR胰岛素抵抗的作用及初步作用机制。方法采用套尾法测定SHR清醒状态下尾动脉收缩压；
Objective:To verify the action effect and elementary mechanism of Clearing Heat and Removing Toxin method on the insulin resistance.
结论增强扫描明显均匀强化，且垂体可见，并出现脑膜尾征以及颈内动脉受推压为鞍内脑膜瘤的特征。
Conclusion Homogeneous enhancement, visibility of the pituitary gland, the dural tail sign and compression of the carotid artery are the characteristics of the intrasellar meningioma.
结论积极行手术切除肿瘤是胰体尾癌肝转移患者获得长期生存的惟一途径，术中肝动脉栓塞化疗是治疗肝转移灶的重要方法。
Conclusion Radical resection is the unique method for long survival and hepatic arterial embolization chemotherapy in operation is an important way for hepatic metastasis.
以胰大动脉为界区分脾动脉近段与远段分别激发有助于胰尾肿瘤的定位。
To separate the proximal splenic artery from the distal one by arteria pancreatic magna and to inject stimulator respectively is helpful for localizing the tumor in pancreas tail.
动物模型制作采用自体动脉血注入大白鼠尾状核，造成脑出血卒中动物模型。
Animal model creation adoption is from the artery blood infuse into white rat's caudal nucleus, and result in the apoplexy the inside of animal model, 0.
结论：大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂，细胞信号转导通路MAPK的激活参与其收缩效应。
CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.
结论：大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂，细胞信号转导通路MAPK的激活参与其收缩效应。
CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.
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