结论SAB对细胞缺糖损伤有明显的保护作用，其作用机制与抑制钙离子内流有关。
Conclusion SAB can protect cells from injury of glucose deprivation and the mechanism is related with its effect of mediating cellular calcium ion.
目的：观察川芎嗪对心肌细胞缺氧缺糖损伤的保护作用。
OBJECTIVE: To investigate the protective effects of ligustrazine on cultural myocardial cells against hypoxia and lack of sugar.
结论：川芎嗪对心肌细胞缺氧缺糖损伤有较好的保护作用。
CONCLUSION: ligustrazine can improve the protective capability of myocardial cells to the injury of hypoxia and lack of sugar.
结论：川芎嗪对心肌细胞缺氧缺糖损伤有较好的保护作用。
CONCLUSION: ligustrazine can improve the protective capability of myocardial cells to the injury of hypoxia and lack of sugar.
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