• 方法体外培养心肌细胞

    METHODS Myocardial cells of neonatal rats were cultured in vitro.

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  • 模型大鼠心肌细胞结构出现明显病理改变

    The obvious pathological changes of cardiomyocyte structure were found in model group.

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  • 检测大鼠心肌细胞凋亡指数应用原位末端标记法

    Myocardial apoptosis index was detected with in situ end labeling.

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  • 大鼠心肌细胞采用离心密度梯度离心分离提纯。

    Velocity and isopyknic gradient centrifugation was employed to fractionate rat myocardial nuclei.

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  • 目的观察和营安心慢性心衰大鼠心肌细胞凋亡影响

    ObjectiveTo study the effects of Heyingansxin Fang on myocardial apoptosis in chronic congestive heart failure rats.

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  • 目的观察生脉胶囊慢性心力衰竭大鼠心肌细胞凋亡影响

    Objective To research the effect of Shengmai capsules on cadiocyte apoptosis with chronic congestive heart failure (CHF) in rats.

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  • 表明SD大鼠心肌细胞条件培养基用于胚胎细胞分离培养

    The essay showed SD rat cardiomyocyte conditioned medium can be used to isolation and culture of rabbit ES-like cell.

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  • 目的探讨糖尿病大鼠心肌细胞肌浆网蛋白基因表达改变。

    AIM: To investigate the gene expression of proteins handling the calcium homeostasis in diabetic rat heart.

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  • 目的观察严重烧伤延迟复苏大鼠心肌细胞线粒体呼吸功能变化

    Objective: to investigate the changes of mitochondria respiratory function of myocardial cell in burnt rats after delayed fluid resuscitation.

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  • 目的观察不同药物干预下,新生大鼠心肌细胞蛋白含量活力关系

    Objective To observe the relationship between protein sythesis and cardiomyocyte viability in neonatal rats.

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  • 结论性高血压心肌细胞凋亡增多,且凋亡增加时间依赖性

    CONCLUSION The cardiomycyte apoptosis in rats with renal hypertension becomes more and the apoptosis increase shows the time dependence.

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  • 目的探讨宁夏产月见体外培养大鼠心肌细胞抑制作用。

    Objective To study the inhibitive effect of evening primrose oil (EPO) on cultured cardiomyocytes hypertrophy in rats.

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  • 结果表明,VD_3可以引起大鼠心肌细胞通透性增造成心肌坏死

    The results showed that VD_3 could raise the permeability of rat cardiac sarclemmas and cause cardiac necrosis.

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  • 目的:建立大鼠心肌细胞分离方法,用于膜片实验记录L型钙离子通道电流

    Objective: to devise a method to isolate calcium-tolerant cardiomyocytes from SD rats for the experiment of patch clamp, and record the currents of L-type calcium channels.

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  • 目的探讨有氧运动联合膳食控制2型糖尿病大鼠心肌细胞凋亡影响及其机制。

    Objective: to explore the effect of aerobic exercise and dietary control on cardiomyocyte apoptosis in type 2 diabetic rats and analyze the changes in other relevant indicators.

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  • 心肌梗死大鼠心肌细胞增殖程度心脏收缩功能呈正性相关,与舒张功能呈性相关。

    Myocyte proliferation is positively correlated with heart systolic function, and negatively correlated with heart diastolic function in chronic heart failure.

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  • 目的研究1,6二磷酸果糖(FDP)阿霉素(adm)导致大鼠心肌细胞凋亡影响

    AIM to study the effect of fructose 1, 6 diphosphate (FDP) on adriamycin (ADM) induced cardiomyocyte apoptosis in rats.

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  • 目的观察异黄酮(SI)大鼠心肌细胞动作电位影响培养心室细胞电流作用。

    Objective To observe the effects of soybean isoflavone (SI) on the action potential in rats cardiomyocytes and the L-type calcium current in cultured neonatal rat ventricular cells.

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  • 目的研究卡维地洛急性心肌梗死(ami)鼠心肌细胞基质(ecm)重塑影响

    Aim: To study the effect of carvedilol on myocardial extracellular matrix (ECM) remodeling in rats after acute myocardial infarction (AMI).

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  • 目的研究蕨麻丁醇部位大鼠心肌细胞缺氧所致超载抑制作用,并探讨其可能机制

    Objectives: To study the inhibition and mechanisms of n-butanol fraction of Potentilla anserine L. against hypoxia -induced calcium overload in myocardial cells.

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  • 方法应用细胞电压箝技术斜坡脉冲程序,测定大鼠心肌细胞稳态电流-电压关系曲线。

    METHODS: The quasi-steady state current-voltage relationship from the isolated rat ventricular myocytes was measured using whole-cell voltage-clamp techniques with a ramp pulse protocol.

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  • 方法:采用SP免疫组化方法主动脉缩窄慢性心衰大鼠心肌细胞动蛋白进行定性半定量分析

    Methods: Actin in cardiac myocytes of rats with chronic heart failure induced by banding of ascending aorta was analysed qualitatively and hemi-quantitatively via SP method.

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  • 结论RNA干扰技术用于抑制心肌细胞KCNJ2基因表达生物起搏器研究提供了一种新的思路和方法。

    Conclusions:RNAi can be used to suppress the expression of the KCNJ2 gene in rat myocardial cells. This experimant provides a new methos for the biological pacemaker study.

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  • 透射电镜观察显示巴因大鼠心肌细胞肿胀,肌纤维断裂Z线消失,线粒体增生肿胀,部分空泡化,细胞胶原纤维增生。

    Myofibrillar fragmentation, swelling of the cardiac myocytes, absence of the Z line, increases of the mitochondria and collagen fibers were found in ouabain group by transmission electron microscopy.

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  • 方法应用细胞膜片技术记录头碱对大鼠心肌细胞动作电位时程作用,分别给予奎尼丁维拉·帕米处理比较二者动作电位时程的影响

    Methods Effects of quinidine and verapamil on the alteration of action potential duration induced by aconitine were studied by use of the whole cell patch clamp techniques in rat ventricular myocytes.

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  • 发挥作用了他们引发心脏病然后注射新的心肌细胞每个移植物都存活了然后完整进入大鼠心脏

    It worked. When they caused heart attacks in the rats and then injected the new heart muscle cells, every graft survived and integrated into the hearts of the rats.

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  • 这种方法临时纤维蛋白凝胶培养大鼠心肌细胞细胞形成组织后去除纤维蛋白凝胶。

    The approach USES a fibrin gel to support rat cardiac cells temporarily, before the fibrin breaks down as the cells organize into tissue.

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  • 目的观察左旋卡尼汀离体大鼠心脏缺血-再灌注损伤的作用心肌细胞能量代谢影响

    Objective: To study the effects of L-carnitine on myocardial ischemia-reperfusion injury and energy metabolism in rats.

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  • 目的观察热休克预处理大鼠缺血再灌注损伤心肌细胞凋亡表达影响。

    Objective to observe whether the heat-shock pretreatment could restrain the expression of apoptosis cells in ischemia-reperfusion myocardium of rats.

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  • 结论严重烧伤大鼠延迟复苏时会加重心肌细胞线粒体呼吸功能损伤

    Conclusion: Delayed fluid resuscitation results in more severe injury of mitochondria respiratory function of myocardial cell in burnt rats.

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